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Restrictions That Influence Avian Leukosis Virus-Induced Lymphoid Leukosis

1986
Infection of one day old White leghorn chicks with avian leukosis virus (ALV) usually results in the development of a bursal- dependent B-cell lymphoma (Purchase and Burmester, 1978). This tumor requires the bursal environment to develop and is characterized by the presence of cell surface immunoglobulin M (IgM) (Cooper et al., 1974).
E H, Humphries, T W, Baba
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5′ avian leukosis virus sequences and osteopetrotic potential

Virology, 1992
Recombinants of Rous-associated virus-0 and Br21 have been used to localize 5' viral sequences that affect the osteopetrotic potential of avian leukosis viruses. Rous-associated virus-0 is a benign subgroup E virus of endogenous origin that does not cause osteopetrosis. Br21 is a constructed subgroup E virus with high osteopetrotic potential.
Robinson, Harriet L.   +5 more
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Embryonic infection with the endogenous avian leukosis virus Rous-associated virus-0 alters responses to exogenous avian leukosis virus infection

Journal of Virology, 1987
We inoculated susceptible chicken embryos with the endogenous avian leukosis virus Rous-associated virus-0 (RAV-0) on day 6 of incubation. At 1 week after hatching, RAV-0-infected and control chickens were inoculated with either RAV-1 or RAV-2, exogenous viruses belonging to subgroups A and B, respectively.
L B, Crittenden   +3 more
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Avian leukosis virus‐receptor interactions

Avian Pathology, 1998
Cellular receptors for subgroups A, B, D and E avian leukosis virus (ALV) have been identified and characterized. The Tva receptor for subgroup A ALV is a member of the low density lipoprotein receptor family of proteins. There is an accumulating body of evidence to suggest that this receptor binds specifically to subgroup A viral envelope (Env ...
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Yellow Fever Vaccine and Avian Leukosis Virus

Annals of Internal Medicine, 1984
Excerpt To the editor: The recommendations of the Immunization Practices Advisory Committee for yellow fever immunization have recently been updated again (1).
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Plaque assay for some strains of avian leukosis virus

Virology, 1972
Abstract Some strains of avian leukosis virus were found to produce plaques at 41 ° in cultures of chick embryo cells which had been fully infected with a temperature-sensitive mutant of Rous sarcoma virus (RSV) which fails to induce morphological transformation at the elevated temperature.
S, Kawai, H, Hanafusa
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Serologic Survey of Man for Avian Leukosis Virus Infection

The Journal of Immunology, 1967
Summary Six current lots of 17 D yellow fever virus vaccine propagated in chicken embryos were found to be contaminated with complement-fixing group antigens of avian leukosis virus. Bloods of 111 individuals vaccinated with 22 different lots of the 17 D vaccine given up to 54 months before drawing a blood specimen were ...
F, Piraino   +2 more
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Occurrence of subgroup J avian leukosis virus in Taiwan

Avian Pathology, 2002
There are three grandparent farms for three different chicken breeds in Taiwan. One of these farms, populated by breast meat yield chickens (yield type), suffered from a severe subgroup J avian leukosis virus (ALV-J) infection in mid-1997. The affected flocks at that farm had a weekly mortality of more than 1% and a 15% drop in egg production.
C-H, Wang, Y-W, Juan
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Host gene control of endogenous avian leukosis virus production

Virology, 1974
Abstract Chick embryo cell supernates or sera of young line 100 chickens, which were susceptible to avian leukosis-sarcoma viruses of subgroup E, contained large amounts of an avian leukosis virus. This virus had biological, chemical and physical properties identical with RAV-O, an endogenous subgroup E virus of the chicken. Line 100 cells homozygous
L B, Crittenden   +3 more
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An avian leukosis virus associated with stocks of rous sarcoma virus

Virology, 1962
Abstract Isolation of a second virus from stocks of the Bryan high-titer strain of Rous sarcoma virus (RSV) is reported. The newly isolated agent, designated as Rous-associated virus (RAV), can be detected in tissue culture, despite its failure to produce discrete cytological alterations, because it interferes with infection and focus formation by ...
H, RUBIN, P K, VOGT
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