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Distinct basal ganglia hyperechogenicity in idiopathic basal ganglia calcification
Movement Disorders, 2010AbstractWe report a 67‐year‐old patient with idiopathic basal ganglia calcification (IBGC). He presented with progressive cognitive impairment, frontal lobe dysfunction, mild leg spasticity, and levodopa (L‐dopa)‐responsive parkinsonism. Transcranial sonography (TCS) revealed marked hyperechogenicity of the basal ganglia and periventricular spaces ...
Norbert, Brüggemann +4 more
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Neurochemical Oscillations in the Basal Ganglia
Bulletin of Mathematical Biology, 2009This work represents an attempt to elucidate the neurochemical processes in the basal ganglia by mathematical modelling. The correlation between neurochemistry and electrophysiology has been used to construct a dynamical system based on the basal ganglia's network structure.
Noori, H., Jäger, W.
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Molecular and Cellular Neuroscience, 1991
All neurotransmitter and hormone receptors that stimulate adenylyl cyclase are thought to do so via the alpha subunit of the guanine nucleotide binding (G) protein G(s). The basal ganglia contain a well-characterized dopamine-stimulated adenylyl cyclase and D1 dopamine receptors coupled to G(s) are thought to mediate this activity.
S L, Drinnan +3 more
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All neurotransmitter and hormone receptors that stimulate adenylyl cyclase are thought to do so via the alpha subunit of the guanine nucleotide binding (G) protein G(s). The basal ganglia contain a well-characterized dopamine-stimulated adenylyl cyclase and D1 dopamine receptors coupled to G(s) are thought to mediate this activity.
S L, Drinnan +3 more
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The language of the basal ganglia
1997A brief historical account of the evolution of nomenclature frequently used in discussing the structure and function of the basal ganglia in health and disease is presented.
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Neurologic Clinics, 2000
The epidemiology, clinical features, pathology, and mechanisms of action of basal ganglia neurotoxins are reviewed. Manganese, cyanide, hydrogen sulfide, methanol, carbon monoxide, 3-nitropropionic acid, MPTP, and annonaceae alkaloids are discussed. The probable mechanism of action for almost all basal ganglia neurotoxins is inhibition of mitochondrial
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The epidemiology, clinical features, pathology, and mechanisms of action of basal ganglia neurotoxins are reviewed. Manganese, cyanide, hydrogen sulfide, methanol, carbon monoxide, 3-nitropropionic acid, MPTP, and annonaceae alkaloids are discussed. The probable mechanism of action for almost all basal ganglia neurotoxins is inhibition of mitochondrial
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Neoplasms of the basal ganglia.
Rays, 1996Neoplasms of the basal ganglia are deep-seated and this markedly impacts on therapy, especially with respect to the surgical approach. However, neoplasms purely involving the basal ganglia are rare: more frequently, basal ganglia are secondarily involved by tumors originating in the cerebral hemispheres, the ventricles and the pineal or suprasellar ...
SIMONETTI L +4 more
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Basal ganglia: Structure and function
Neuroscience and Behavioral Physiology, 1998Noting considerable successes in the development of studies on the structure and functions of the basal ganglia (as discussed at the most recent Congress), we naturally cannot see a number of major shortcomings of this process. In particular, there is concern regarding the methodological level of investigations.
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The basal ganglia control the detailed kinematics of learned motor skills
Nature Neuroscience, 2021Ashesh Dhawale +2 more
exaly