Results 321 to 330 of about 2,670,501 (370)

Symposium. Brain edema.

Neurologia medico-chirurgica, 1969
K, Ueki, K, Okada, M, Hayano, R, Tanaka, H, Hatanaka   +4 more
openaire   +1 more source

Aquaporins in brain edema

Journal of Neuroscience Research, 2018
AbstractBrain edema is a common feature of brain injuries, which leads to increased intracranial pressure (ICP) and ischemia that worsen outcome. Current management of edema focuses on reduction of ICP, but there are no treatments targeting the molecular players directly involved in edema process.
Tifenn Clément, Beatriz Rodriguez‐Grande, Jérôme Badaut   +2 more
openaire   +3 more sources

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Brain Edema

Diffusion-Weighted MR Imaging of the Brain, Head and Neck, and Spine, 2021
Toshio Moritani, Badih Junior Daou, Gregory Palmateer, Aditya S. Pandey   +3 more
openaire   +2 more sources

Improving the Function of Meningeal Lymphatic Vessels to Promote Brain Edema Absorption after Traumatic Brain Injury

Journal of Neurotrauma, 2022
Brain edema is the most common and fatal complication after traumatic brain injury (TBI). Meningeal lymphatic vessels (MLVs) are the conduits that transport cerebrospinal fluid (CSF) and macromolecules to deep extracranial cervical lymph nodes (dCLNs ...
Jian Liao, M. Zhang, Zichuan Shi, Hao Lu, Li-Chen Wang, Weijia Fan, Xiaoguang Tong, Hua Yan   +7 more
semanticscholar   +1 more source

Overexpression of long noncoding RNA Malat1 ameliorates traumatic brain injury induced brain edema by inhibiting AQP4 and the NF‐κB/IL‐6 pathway

Journal of Cellular Biochemistry, 2019
Brain edema is a major traumatic brain injury (TBI)‐related neurological complication. In the initiation stage of TBI, brain edema is characterized by astrocyte swelling (cytotoxic edema). We studied the impact of a long noncoding RNA, Malat1, on the TBI‐
Yamin Zhang, Jianping Wang, Yi Zhang, Jia Wei, Ruipeng Wu, H. Cai   +5 more
semanticscholar   +1 more source

Glymphatic System Impairment Contributes to the Formation of Brain Edema After Ischemic Stroke

Stroke
BACKGROUND: Blood-brain barrier damage has traditionally been considered to determine the occurrence and development of poststroke brain edema, a devastating and life-threatening complication.
Juan Zhu, Jiaying Mo, Kewei Liu, Quanfeng Chen, Zheqi Li, Yihua He, Yuan Chang, Chuman Lin, Mingjia Yu, Yikai Xu, Xiangliang Tan, Kaibin Huang, Suyue Pan   +12 more
semanticscholar   +1 more source

Ischemic brain edema

Progress in Cardiovascular Diseases, 1999
Brain edema is a life-threatening complication of cerebral infarction. The molecular cascade initiated by cerebral ischemia includes the loss of membrane ionic pumps and cell swelling. Secondary formation of free radicals and proteases disrupts brain-cell membranes, causing irreversible damage. New diagnostic methods based on magnetic resonance imaging
openaire   +2 more sources

Brain Edema

1986
Current concepts on the development of brain edema are briefly reviewed. The common classification into vasogenic and cytotoxic edema is a simplification and does not cover all types of edema. Alternative routes of edema resolution are discussed. More basic data on the mechanisms of development and regression of brain edema are needed before more ...
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[Brain edema].

Therapeutische Umschau. Revue therapeutique, 2005
The most frequent complication of an acute cerebral lesion is brain edema. With increasing size of brain edema an increase in intracranial pressure is observed. This leads to decreased cerebral perfusion and brain death. Cerebral edema can be subdivided into three groups: vasogenic, cytotoxic and interstitial brain edema.
openaire   +3 more sources