Results 181 to 190 of about 570,584 (333)
Editorial: Unlocking potential: cognitive rehabilitation for individuals with acquired brain injuries. [PDF]
Vas AK +4 more
europepmc +1 more source
Diffusion‐Weighted Imaging for the Evaluation of the Sacroiliac Joint in Pediatric Patients
Background Maturational signal in the sacroiliac joint (SIJ) of skeletally immature youth is often misinterpreted as inflammation. Diagnostic tools that improve specificity are greatly needed. Apparent diffusion coefficient (ADC) values from diffusion‐weighted imaging (DWI), when used with standard imaging, may enhance diagnostic accuracy.
Michael L. Francavilla +6 more
wiley +1 more source
Proteins are New Molecular Markers When Analyzing Material Structures to Prevent Traumatic Brain Injuries [PDF]
von Holst H +4 more
openalex +1 more source
Objective This study investigates the association between discordance in commonly collected clinical indicators of rheumatoid arthritis (RA) disease activity and abnormalities in quantitative sensory testing (QST) observed in individuals with nociplastic pain.
Clarice P. Lin +10 more
wiley +1 more source
Stem Cell-Derived Extracellular Vesicle Therapy in Ischemic Brain Injuries. [PDF]
Qiu C, Lindley A, Jia X.
europepmc +1 more source
Mechanisms of Secondary Neuronal Damage in Severe Brain Injury (Part 2)
V. V. Moroz +2 more
openalex +2 more sources
Objective A patient‐centered approach for chronic disease management, including systemic lupus erythematosus (SLE), aligns treatment with patients’ values and preferences, leading to improved outcomes. This paper summarizes how patient experiences, perspectives, and priorities informed the American College of Rheumatology (ACR) 2024 Lupus Nephritis (LN)
Shivani Garg +20 more
wiley +1 more source
Beyond Inflammation: Why Understanding the Brain Matters in Inflammatory Arthritis
Persistent pain remains a major challenge in inflammatory arthritis, even when joint inflammation is well controlled. Pain and associated symptoms such as fatigue cannot be explained by peripheral inflammation alone but reflect altered central pain processing. These changes may arise through “top‐down” mechanisms, reflecting pre‐existing dysfunction in
Eoin M. Kelleher +2 more
wiley +1 more source

