Results 301 to 310 of about 358,469 (348)
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European Neurology, 1981
Changes in the maximal rate of some cerebral enzymatic activities related to 400ene transduction and neurotransmission (lactate dehydrogenase; citrate synthase and malate dehydrogenase; total NADH-cytochrome c reductase and cytochrome oxidase; glutamate dehydrogenase; acetylcholine esterase) were assayed both in the crude or purified mitochondrial ...
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Changes in the maximal rate of some cerebral enzymatic activities related to 400ene transduction and neurotransmission (lactate dehydrogenase; citrate synthase and malate dehydrogenase; total NADH-cytochrome c reductase and cytochrome oxidase; glutamate dehydrogenase; acetylcholine esterase) were assayed both in the crude or purified mitochondrial ...
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Revue Neurologique, 2011
The extent of the penumbra and the core of an acute ischemic stroke influence, at the given time, the impact of the recanalization of the occluded vessel on the outcome. Research studies have demonstrated that quantitative MR diffusion imaging and, to a lesser extent, CT perfusion (CTP) could provide an acceptable estimation of the size of the core ...
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The extent of the penumbra and the core of an acute ischemic stroke influence, at the given time, the impact of the recanalization of the occluded vessel on the outcome. Research studies have demonstrated that quantitative MR diffusion imaging and, to a lesser extent, CT perfusion (CTP) could provide an acceptable estimation of the size of the core ...
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The pathophysiology of brain ischemia
Annals of Neurology, 1983AbstractBrain ischemia due to a critical reduction in cerebral blood flow is a well recognized and common cause of irreversible brain damage. The observation that brain cells are more resistant to ischemia than was previously assumed on the basis of clinical experience has stimulated considerable investigative work designed to determine those factors ...
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Cerebral ischemia in the developing brain
Journal of Cerebral Blood Flow & Metabolism, 2022Brain ischemia affects all ages, from neonates to the elderly population, and is a leading cause of mortality and morbidity. Multiple preclinical rodent models involving different ages have been developed to investigate the effect of ischemia during different times of key brain maturation events. Traditional models of developmental brain ischemia have
Robert M Dietz +2 more
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Diabetes, 1996
Diabetes influences brain ischemia in a number of different ways. Diabetes causes and exacerbates macroangiopathies, increases the severity of ischemia, and increases stroke mortality. Unfortunately, few studies have examined in sufficient depth the influence of diabetes on the various vascular lesions that cause brain ischemia.
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Diabetes influences brain ischemia in a number of different ways. Diabetes causes and exacerbates macroangiopathies, increases the severity of ischemia, and increases stroke mortality. Unfortunately, few studies have examined in sufficient depth the influence of diabetes on the various vascular lesions that cause brain ischemia.
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Hyperthermia and Brain Ischemia
1997This chapter reviews recent experimental studies concerning the detrimental consequences of hyperthermia on ischemic outcome. The chapter also discusses potential mechanisms underlying the detrimential consequences of mild hyperthermia. Fever is frequently observed following cardiopulmonary resuscitation and is considered prognostic of poor outcome in ...
Raul Busto, W. Dalton Dietrich
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1995
Stroke is the second or third major cause of death in the “industrialized countries.” About 30% of patients die within a year after the first insult, and 20% remain dependent on care. So far there is no effective therapy available but one is desperately needed.
Karl Rudolphi, Peter Schubert
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Stroke is the second or third major cause of death in the “industrialized countries.” About 30% of patients die within a year after the first insult, and 20% remain dependent on care. So far there is no effective therapy available but one is desperately needed.
Karl Rudolphi, Peter Schubert
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Neurogenesis following brain ischemia
Developmental Brain Research, 2002Following 5 or 10 min of global ischemia in the adult gerbil there is a tenfold increase in the birth of new cells in the subgranular zone of dentate gyrus of the hippocampus as assessed using BrdU incorporation. This begins at 7 days, peaks at 11 days, and decreases thereafter. Over the next month approximately 25% of the newborn cells disappear.
Ramon Bernabeu +2 more
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Catecholamines in Experimental Brain Ischemia
Archives of Neurology, 1975Local cerebral ischemia was produced in rats by internal carotid artery injection of 35 mu carbon microspheres, and brain norepinephrine (NE), dopamine, and cyclic adenosine 3, 5-monophosphate (cAMP) were measured in embolized and intact hemispheres at intervals up to four hours. Sham-operated animals were controls.
Kyuya Kogure +4 more
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Inflammation of the Brain after Ischemia
1996Cytokines which promote emigration of leukocytes from the vascular lumen into the injured brain tissue are produced at the site of incipient cerebral infarction. The blood-borne invaders then accelerate the decomposition of brain cells by their toxic by-products, phagocytic action, and by the immune reaction.
Kyuya Kogure +4 more
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