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Vascular Protection in Brain Ischemia
Cerebrovascular Diseases, 2006Vascular damage occurring after cerebral ischemia may lead to a worse outcome in patients with ischemic stroke, as it facilitates edema formation and hemorrhagic transformation. There are several phases in the development of vascular injury (acute, subacute and chronic) and different mediators act in each one.
Mar Castellanos +4 more
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Global Brain Ischemia and Reperfusion
Annals of Emergency Medicine, 1996Brain damage accompanying cardiac arrest and resuscitation is frequent and devastating. Neurons in the hippocampus CA1 and CA4 zones and cortical layers III and V are selectively vulnerable to death after injury by ischemia and reperfusion. Ultrastructural evidence indicates that most of the structural damage is associated with reperfusion, during ...
Donald J. DeGracia +6 more
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Cerebral ischemia and brain histamine
Brain Research Reviews, 2005Cerebral ischemia induces excess release of glutamate and an increase in the intracellular Ca(2+) concentration in neurons, which provokes enzymatic process leading to irreversible neuronal injury. Histamine plays a role as a neurotransmitter in the mammalian brain, and histamine release from nerve endings is enhanced in ischemia by facilitation of ...
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1977
Complete cessation of cerebral blood flow causes an almost immediate suppression of higher central nervous function (41), followed by a breakdown of cell metabolism within a few minutes (36, 37). The biochemical and functional sequel of ischemic suppression has been studied extensively in various laboratories, and the results obtained are quite similar
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Complete cessation of cerebral blood flow causes an almost immediate suppression of higher central nervous function (41), followed by a breakdown of cell metabolism within a few minutes (36, 37). The biochemical and functional sequel of ischemic suppression has been studied extensively in various laboratories, and the results obtained are quite similar
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Experimental ischemia of the brain
1977Cerebral ischemia as seen by the vascular surgeon generally is the consequence of regional stenosis or occlusion of the main supply vessels of the brain. The pathophysiology of regional ischemia is a very complex one: areas of no-flow or low-flow are surrounded by regions of reactive hyperemia, causing different degrees of ischemic cell damage. Changes
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Gangliosides in brain ischemias
Neurologia Croatica, 1994The aim of this biochemical study was to explore the changes of gangliosides in brain tissue affected by ischemic damage. Samples of brain tissue with ischemic lesion (grey and white matter) (n=5), tissue adjacent to lesion (grey and white matter)(n=3) and contralateral control tissue (grey and white matter)(n=5) were analyzed.
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Brain Ischemia and Neuronal Excitability
2009Selective neuronal death in certain brain regions has long been recognized as a consequence of transient cerebral ischemia; however, its mechanisms remain unclear. Growing evidence indicates that an increase in neuronal excitability may contribute to this process.
Zao C. Xu, Ping Deng
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Thrombin induces ACSL4-dependent ferroptosis during cerebral ischemia/reperfusion
Signal Transduction and Targeted Therapy, 2022Yang Shu, Yu-Jie Guo, Scott Ayton
exaly
[Balneotherapy of brain ischemia].
Voprosy kurortologii, fizioterapii, i lechebnoi fizicheskoi kultury, 2004F K Balakishieva, Musaev Av
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