Results 11 to 20 of about 18,700 (219)

All that looks like “Brugada” is not “Brugada”: Case series of Brugada phenocopy caused by hyponatremia

open access: yesJournal of the Saudi Heart Association, 2016
Brugada syndrome (BS), a life-threatening channelopathy associated with reduced inward sodium current due to dysfunctional sodium channels, is characterized by ST-segment elevation with downsloping “coved type” (type 1) or “saddle back” (type 2) pattern ...
Yashwant Agrawal   +3 more
doaj   +4 more sources

Brugada syndrome, Brugada phenocopy or none? [PDF]

open access: yesAnnals of Noninvasive Electrocardiology, 2017
Brugada syndrome is a form of inherited arrhythmia syndrome characterized by a distinctST‐segment elevation in the right precordial leads. Brugada phenocopies are clinical entities that present with an electrocardiographic pattern identical to Brugada syndrome and may obey to various clinical conditions.
Umut Kocabas   +4 more
openaire   +3 more sources

Suspected hyponatremia-induced Brugada phenocopy

open access: yesInternational Medical Case Reports Journal, 2019
Koomatie Ramsaroop,1 Rajeev Seecheran,1 Valmiki Seecheran,1 Sangeeta Persad,1 Stanley Giddings,2 Boris Mohammed,1 Naveen Anand Seecheran2 1Department of Medicine, North Central Regional Health Authority, Mt Hope, Trinidad and Tobago; 2Department of ...
Ramsaroop K   +6 more
doaj   +1 more source

WHEN “BRUGADA HEART” STOPS BEATING [PDF]

open access: yesРоссийский кардиологический журнал, 2012
The typical Brugada patient is young, otherwise healthy male with normal general medical and cardiovascular physical findings. A present a case of successful resuscitation following “Brugada heart” arrest, where diagnosis of type 2 Brugada syndrome was ...
Andjelic Sladjana
doaj   +1 more source

Brugada syndrome [PDF]

open access: yesActa Cardiologica, 2020
Brugada syndrome (BrS) is an inherited cardiac arrhythmia syndrome that causes a heightened risk for ventricular tachyarrhythmias and sudden cardiac death. BrS is characterised by a coved ST-segment elevation in right precordial leads. The prevalence is estimated to range between 1 in 5,000 to 1 in 2,000 in different populations, with the highest being
Korlipara, Haarika   +2 more
openaire   +2 more sources

Prevalence of Brugada-type ECG pattern and early ventricular repolarization pattern in Tunisian athletes

open access: yesOpen Access Journal of Sports Medicine, 2011
Sana Ouali1, Helmi Ben Salem1, Sami Hammas1, Elyes Neffeti1, Fahmi Remedi1, Abdallah Mahdhaoui2, Essia Boughzela1, Rafik Mankai31Department of Cardiology, Sahloul Hospital, Sousse, Tunisia; 2Department of Cardiology, Farhat Hached, Sousse, Tunisia ...
Ouali S   +7 more
doaj   +1 more source

Brugada Syndrome

open access: yesJACC: Clinical Electrophysiology, 2022
Brugada syndrome (BrS) is an "inherited" condition characterized by predisposition to syncope and cardiac arrest, predominantly during sleep. The prevalence is ∼1:2,000, and is more commonly diagnosed in young to middle-aged males, although patient sex does not appear to impact prognosis.
Krahn, AD   +5 more
openaire   +2 more sources

Brugada syndrome [PDF]

open access: yesOrphanet Journal of Rare Diseases, 2006
A novel clinical entity characterized by ST segment elevation in right precordial leads (V1 to V3), incomplete or complete right bundle branch block, and susceptibility to ventricular tachyarrhythmia and sudden cardiac death has been described by Brugada et al. in 1992. This disease is now frequently called "Brugada syndrome" (BrS).
Napolitano C, PRIORI, SILVIA GIULIANA
openaire   +6 more sources

Featuring: Josep Brugada [PDF]

open access: yes, 2019
In the Cardiology Masters section of European Cardiology Review, we bring you an insight into the career of a key contributor to the field of cardiology.
Josep Brugada
core   +1 more source

Phase 2 Re-Entry Without Ito: Role of Sodium Channel Kinetics in Brugada Syndrome Arrhythmias. [PDF]

open access: yes, 2023
BACKGROUND: In Brugada syndrome (BrS), phase 2 re-excitation/re-entry (P2R) induced by the transient outward potassium current (Ito) is a proposed arrhythmia mechanism; yet, the most common genetic defects are loss-of-function sodium channel mutations ...
Qu, Zhilin   +4 more
core   +1 more source

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