Results 261 to 270 of about 3,468,317 (412)
XXXIV.—Solubilities of the calcium salts of the acids of the acetic series [PDF]
John S. Lumsden
openalex +1 more source
Exploration of heterogeneity and recurrence signatures in hepatocellular carcinoma
This study leveraged public datasets and integrative bioinformatic analysis to dissect malignant cell heterogeneity between relapsed and primary HCC, focusing on intercellular communication, differentiation status, metabolic activity, and transcriptomic profiles.
Wen‐Jing Wu+15 more
wiley +1 more source
Secular trends of vitamin D and calcium intake and their circulating levels in US adults from 2007 to 2018. [PDF]
Du Y+7 more
europepmc +1 more source
TGF‐β has a complex role in cancer, exhibiting both tumor‐suppressive and tumor‐promoting properties. Using a series of differentiated tumoroids, derived from different stages and mutational background of colorectal cancer patients, we replicate this duality of TGF‐β in vitro. Notably, the atypical but highly aggressive KRASQ22K mutation rendered early‐
Theresia Mair+17 more
wiley +1 more source
Dysregulation of STS in keratinocytes promotes calcium signaling and differentiation. [PDF]
Kwon TU+4 more
europepmc +1 more source
CALCIUM METABOLISM IN A CASE OF GARGOYLISM, STUDIED WITH THE AID OF RADIOCALCIUM1 [PDF]
Félix Bronner+3 more
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Germline variants in CDKN2A wild‐type melanoma prone families
Among melanoma‐prone families, wild‐type for CDKN2A and CDK4, some have pathogenic variants in genes not usually linked to melanoma. Furthermore, rare XP‐related variants and variants in MC1R are enriched in such families. Germline pathogenic variants in CDKN2A are well established as an underlying cause of familial malignant melanoma. While pathogenic
Gjertrud T. Iversen+5 more
wiley +1 more source
Evidence of bacterial imprints in different types of non-struvite kidney stones. [PDF]
Grases F+7 more
europepmc +1 more source
Chronic TGF‐β exposure drives epithelial HCC cells from a senescent state to a TGF‐β resistant mesenchymal phenotype. This transition is characterized by the loss of Smad3‐mediated signaling, escape from senescence, enhanced invasiveness and metastatic potential, and upregulation of key resistance modulators such as MARK1 and GRM8, ultimately promoting
Minenur Kalyoncu+11 more
wiley +1 more source