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Calorie restriction induces mandible bone loss by regulating mitochondrial function. [PDF]
Liu L +7 more
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Brain response to calorie restriction
Cellular and Molecular Life Sciences, 2012Calorie restriction extends longevity and delays ageing in model organisms and mammals, opposing the onset and progression of an array of age-related diseases. These beneficial effects also extend to the maintenance of brain cognitive functions at later age and to the prevention, at least in rodents, of brain senescence and associated neurodegenerative
Fusco, Salvatore, Pani, Giovambattista
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Experimental Gerontology, 1997
Current data suggest that the life spans of commonly held rodent species have decreased and the occurrence of tumors occurs earlier in the lifespan of ad libitum-fed animals. The most likely cause of this change in the life span of barrier-reared genetically defined animals is increased body weight.
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Current data suggest that the life spans of commonly held rodent species have decreased and the occurrence of tumors occurs earlier in the lifespan of ad libitum-fed animals. The most likely cause of this change in the life span of barrier-reared genetically defined animals is increased body weight.
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Antiaging, longevity and calorie restriction
Current Opinion in Clinical Nutrition and Metabolic Care, 2010The role of calorie restriction in humans is controversial. Recently, new data in monkeys and humans have provided new insights into the potential role of calorie restriction in longevity.A study in rhesus monkeys showed a reduction in aging-associated mortality.
John E, Morley +2 more
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Sirtuin regulation in calorie restriction
Biochimica et Biophysica Acta (BBA) - Proteins and Proteomics, 2010The beneficial effects of calorie restriction diet in extending lifespan and preventing diseases have long been recognized. Recent genetic and molecular studies in model organisms began to uncover the molecular regulation of calorie restriction response, with the gene SIR2 playing an essential role. This article summarizes the latest development on how
Xiaolei, Qiu +3 more
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1989
Calorie restriction stresses the impaired hepatocyte transport defect for unconjugated bilirubin in Gilbert’s syndrome, in which there is intracellular endoplasmic UDP glucuronyl transferase deficiency. As a result there is an excessive rise in both serum unconjugated and total bilirubin.
P. T. Lascelles, D. Donaldson
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Calorie restriction stresses the impaired hepatocyte transport defect for unconjugated bilirubin in Gilbert’s syndrome, in which there is intracellular endoplasmic UDP glucuronyl transferase deficiency. As a result there is an excessive rise in both serum unconjugated and total bilirubin.
P. T. Lascelles, D. Donaldson
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