Results 211 to 220 of about 4,784,374 (307)
Arginine methylation can be viewed as a persistence‐prone post‐translational modification regulated by a network of PRMTs. Competitive and compensatory interactions among PRMTs can redistribute methylation across substrate pools shaped by sequence, structural, spatial, and environmental layers, reinforcing RNA‐processing, chromatin, and signaling ...
So Hyun Kwon, Ji Min Lee
wiley +1 more source
Cost-Effectiveness of Smoking Cessation Interventions Integrated Into Lung Cancer Screening.
Cao P +13 more
europepmc +1 more source
Artificial Intelligence as an Assistive Tool in Breast Cancer Screening and Diagnosis in the Digital Era. [PDF]
Rana MK +6 more
europepmc +1 more source
Mechanisms and therapeutic opportunities of the ribotoxic stress response in cancer
Cancer cells' high translational demand creates opportunities to therapeutically target ribosome function. Ribosome stalling and collisions activate ZAKα and the ribotoxic stress response (RSR), which can trigger rapid, p53‐independent apoptosis in cancer.
Anastassiya Kim +7 more
wiley +1 more source
Women's compliance with cervical cancer screening recommendations and associated barriers to participation: A cross-sectional analysis. [PDF]
Spriņģe L +9 more
europepmc +1 more source
This study integrates publicly available transcriptomic datasets to identify molecular signatures associated with response to neoadjuvant chemoradiotherapy in locally advanced rectal cancer. By analyzing a combination of multiple cohorts with bioinformatics approaches, we reveal biological pathways and immune‐related features that may improve ...
Aleksandra Stanojevic +10 more
wiley +1 more source
SNAP Participation and Cancer Screening for Adults With Food Insecurity.
Mevawalla A +8 more
europepmc +1 more source
Prostate Cancer Screening: Current Controversies and Future Directions. [PDF]
Buali HF +7 more
europepmc +1 more source
The VHL tumor suppressor at the crossroad of protein folding, aggregation, and cancer
Mutations, environmental stress, and chaperone dysfunction can destabilize pVHL, promoting its conversion from the native folded state into amyloid‐like assemblies. This transition may contribute to protein storage, cell dormancy, survival, and drug resistance.
Lara Abad +2 more
wiley +1 more source

