Results 41 to 50 of about 120,376 (304)

2D Echocardiographic features in low T3 syndrome in chronic heart failure

open access: yesAsian Journal of Medical Sciences, 2014
Background: Thyroid abnormalities are common in chronic heart failure. Severity of heart failure rises by several fold in patients with thyroid dysfunction.
Arun Kumar
doaj   +1 more source

Anticoagulation in Dilated Cardiomyopathy

open access: yesJournal of the American College of Cardiology, 1998
Patients with dilated cardiomyopathy have multiple factors that predispose to thromboembolic events. However, reports of the incidence of thromboembolic events in this population vary widely. There has never been a controlled study of long-term anticoagulation among patients with congestive heart failure due to dilated cardiomyopathy. In this report we
Lauren S. Koniaris, Samuel Z. Goldhaber
openaire   +2 more sources

Selecting patients with nonischemic dilated cardiomyopathy for ICDs [PDF]

open access: yes, 2017
No abstract ...
Cleland, John G.F.   +2 more
core   +1 more source

Automated identification and quantification of myocardial inflammatory infiltration in digital histological images to diagnose myocarditis [PDF]

open access: yesarXiv, 2023
This study aims to develop a new computational pathology approach that automates the identification and quantification of myocardial inflammatory infiltration in digital HE-stained images to provide a quantitative histological diagnosis of myocarditis.898 HE-stained whole slide images (WSIs) of myocardium from 154 heart transplant patients diagnosed ...
arxiv  

Hypertrophic cardiomyopathy is associated with dilated sinus of Valsalva: A case-control study

open access: yesInternational Journal of Cardiology: Heart & Vasculature, 2023
Background: We aimed to test the hypothesis that there is an association between hypertrophic cardiomyopathy and dilated aorta in a case-control, matched-design fashion. Methods: Of 65,843 studies done from November 2011 to December 2015, we found, after
Ji Ae Yoon   +6 more
doaj  

High-Throughput Precision Phenotyping of Left Ventricular Hypertrophy with Cardiovascular Deep Learning [PDF]

open access: yes, 2021
Left ventricular hypertrophy (LVH) results from chronic remodeling caused by a broad range of systemic and cardiovascular disease including hypertension, aortic stenosis, hypertrophic cardiomyopathy, and cardiac amyloidosis. Early detection and characterization of LVH can significantly impact patient care but is limited by under-recognition of ...
arxiv   +1 more source

Novel compound mutations in the mitochondrial translation elongation factor (TSFM) gene cause severe cardiomyopathy with myocardial fibro-adipose replacement [PDF]

open access: yes, 2019
Primary mitochondrial dysfunction is an under-appreciated cause of cardiomyopathy, especially when cardiac symptoms are the unique or prevalent manifestation of disease.
Bonnen, Penelope E   +16 more
core   +2 more sources

Apoptosis in Dilated Cardiomyopathy

open access: yesThe Korean Journal of Internal Medicine, 2000
Cardiomyopathy, a popular diagnosis that always obscures more than it reveals, nevertheless has several characteristic histological features. These prominently include widespread focal myocardial fibrosis and associated hypertrophy of surviving cardiac myocyte.
Bum-Kee Hong   +10 more
openaire   +3 more sources

Neuraminidase 1 Exacerbated Glycolytic Dysregulation and Cardiotoxicity by Destabilizing SIRT1 through Interactions with NRF2 and HIF1α

open access: yesAdvanced Science, EarlyView.
NEU1, a key regulator of glycolysis, is markedly upregulated following DOX treatment. This upregulation is attributed to HIF1α’s transcriptional repression, requiring intricate interactions with NRF2. Increased NEU1 facilitates SIRT1 lysosomal degradation, contributing to aberrant glycolytic phenotype and cardiac damage.
Ting Gao   +13 more
wiley   +1 more source

Left ventricular non-compaction: clinical features and cardiovascular magnetic resonance imaging [PDF]

open access: yes, 2009
Background: It is apparent that despite lack of family history, patients with the morphological characteristics of left ventricular non-compaction develop arrhythmias, thrombo-embolism and left ventricular dysfunction.
A Keren   +32 more
core   +5 more sources

Home - About - Disclaimer - Privacy