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Science, 1996
Letters from: [ Jan Novak ][1] [ Alice B. Fulton ][1] [ Jean Claude Ameisen ][1] Jean Claude Ameisen's Perspective “The origin of programmed cell death [PCD]” ([31 May, p. 1278][2]) discusses possible evolutionary roots of cell suicide.
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Letters from: [ Jan Novak ][1] [ Alice B. Fulton ][1] [ Jean Claude Ameisen ][1] Jean Claude Ameisen's Perspective “The origin of programmed cell death [PCD]” ([31 May, p. 1278][2]) discusses possible evolutionary roots of cell suicide.
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Clinical Pharmacology & Therapeutics, 2007
Functional consequences of myocardial or cerebral infarction are the result of excessive cell death. It is patent that preventing cell death is the therapeutic goal in any ischemia-reperfusion setting. Mitochondria amplify apoptotic cascades and have emerged as crucial organelles in ischemia-reperfusion.
Hausenloy DJ, SCORRANO, LUCA
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Functional consequences of myocardial or cerebral infarction are the result of excessive cell death. It is patent that preventing cell death is the therapeutic goal in any ischemia-reperfusion setting. Mitochondria amplify apoptotic cascades and have emerged as crucial organelles in ischemia-reperfusion.
Hausenloy DJ, SCORRANO, LUCA
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Transplantation, 1998
Programmed cell death (PCD) is currently one of the most intensively studied areas in cell biology. Substantial evidence now exists demonstrating the integral role of PCD in many fundamental immunologic processes; therefore, understanding the mechanisms of PCD may provide advances with broad implications in immunobiology.
E S, Woodle, S, Kulkarni
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Programmed cell death (PCD) is currently one of the most intensively studied areas in cell biology. Substantial evidence now exists demonstrating the integral role of PCD in many fundamental immunologic processes; therefore, understanding the mechanisms of PCD may provide advances with broad implications in immunobiology.
E S, Woodle, S, Kulkarni
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Journal of Neurobiology, 1992
AbstractExcitotoxicity refers to the ability of glutamate or related excitatory amino acids to mediate the death of central neurons under certain conditions, for example, after intense exposure. Such excitotoxic neuronal death may contribute to the pathogenesis of brain or spinal cord injury associated with several human disease states.
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AbstractExcitotoxicity refers to the ability of glutamate or related excitatory amino acids to mediate the death of central neurons under certain conditions, for example, after intense exposure. Such excitotoxic neuronal death may contribute to the pathogenesis of brain or spinal cord injury associated with several human disease states.
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Nature Reviews Immunology, 2018
The long-term survival of HIV-1-infected CD4+ T cells is shown to be controlled by the anti-apoptotic protein BIRC5 and its upstream regulator OX40, which suggests new therapeutic targets to reduce the size of the viral reservoir.
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The long-term survival of HIV-1-infected CD4+ T cells is shown to be controlled by the anti-apoptotic protein BIRC5 and its upstream regulator OX40, which suggests new therapeutic targets to reduce the size of the viral reservoir.
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Cytotechnology, 1991
The type of DNA damage and the role of poly (ADP-ribosyl) polymerase (ADPRP) and sulphated glyprotein 2 (SGP-2) in programmed cell death (apoptosis) was investigated in the following model systems: i) rat thymocytes treated with dexamethasone (DEX) eitherin vitro orin vivo; ii) human perypheral blood mononuclear cells (hPBMCs) exposed to oxygen free ...
MONTI, DANIELA +8 more
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The type of DNA damage and the role of poly (ADP-ribosyl) polymerase (ADPRP) and sulphated glyprotein 2 (SGP-2) in programmed cell death (apoptosis) was investigated in the following model systems: i) rat thymocytes treated with dexamethasone (DEX) eitherin vitro orin vivo; ii) human perypheral blood mononuclear cells (hPBMCs) exposed to oxygen free ...
MONTI, DANIELA +8 more
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Science, 1997
Cells can die in two ways: in a disorderly, destructive process called necrosis or by programmed cell death (or apoptosis), an orderly series of biochemical events that neatly eliminate the cell. Four reports in this week's issue by Chinnaiyan ( 1122 ), Wu ( 1126 ), Yang (p. 1129), and Kluck (
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Cells can die in two ways: in a disorderly, destructive process called necrosis or by programmed cell death (or apoptosis), an orderly series of biochemical events that neatly eliminate the cell. Four reports in this week's issue by Chinnaiyan ( 1122 ), Wu ( 1126 ), Yang (p. 1129), and Kluck (
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Methods in enzymology, 2008
Granzymes are cell death-inducing serine proteases released from cytotoxic granules of cytotoxic T lymphocytes and natural killer cells during granule exocytosis in response to viral infection or against transformed cells marked for elimination. A critical cofactor for the granule exocytosis pathway is perforin, which mediates the entry of granzymes ...
Martinvalet, Denis +2 more
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Granzymes are cell death-inducing serine proteases released from cytotoxic granules of cytotoxic T lymphocytes and natural killer cells during granule exocytosis in response to viral infection or against transformed cells marked for elimination. A critical cofactor for the granule exocytosis pathway is perforin, which mediates the entry of granzymes ...
Martinvalet, Denis +2 more
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