Results 271 to 280 of about 2,032,190 (314)

Calmodulin and cell proliferation

Biochemical and Biophysical Research Communications, 1982
Abstract The calmodulin content of synchronized Chinese hamster ovary (CHO-K 1 ) cells was determined at each phase of the cell cycle. The calmodulin content was minimum in the G 1 phase, increased after the cells entered S phase and reached the maximum level at the late G 2 or early M phase.
Y, Sasaki, H, Hidaka
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Cell proliferation on hydrogels

In Vitro Cellular & Developmental Biology, 1990
The adhesion and proliferation of mammalian fibroblasts (Flow 7000) on the surface of hydrophilic (copolymer of N-vinyl-2-pyrrolidone and methyl methacrylate) and hydrophobic [polymethylmethacrylate (PMMA) stereocomplex] hydrogels with a wide range in water content were studied morphologically and quantitatively.
S, Nagaoka, H, Tanzawa, J, Suzuki
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Cell proliferation in cancer

European Journal of Cancer (1965), 1968
Resume Dans tous les cas de tumeurs solides etudies in vivo , le temps de doublement cellulaire ( t c ) est plus court que dans les tissus normaux correspondants, en etat d'equilibre. Le temps de doublement des cellules leucemiques blastiques est approximativement le meme que celui des cellules medullaires normales correspondantes, et, a l'heure ...
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Phosphoinositides and cell proliferation

Journal of Cell Science, 1985
ABSTRACT Certain growth factors act by stimulating the hydrolysis of inositol lipids to yield putative second messengers such as diacylglycerol (DG) and inositol trisphosphate (IP3). One function of the former is to stimulate C-kinase, which may act by switching on a sodium/hydrogen exchanger to induce the increase in pH that appears to ...
M J, Berridge, K D, Brown, R F, Irvine, J P, Heslop   +3 more
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Cell Proliferation and Carcinogenesis

Drug Metabolism Reviews, 1998
(1998). Cell Proliferation and Carcinogenesis. Drug Metabolism Reviews: Vol. 30, No. 2, pp. 339-357.
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Proliferation: the Cell Cycle

2003
Normal cells possess mechanisms that protect them from the action of oncogenes. Thus, normal primary cells are not affected by neoplastic transformations, whereas the majority of immortal cells used in laboratory cultures have acquired mutations during the immortalization process and are susceptible to being transformed.
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Cell proliferation and control

Current Biology, 1992
In the past year much of the focus in cell-cycle research has turned from the regulation of mitosis to the control of the initiation of DNA replication. Novel findings include the discovery of vertebrate G1 cyclins, an additional cdc2-related kinase potentially involved in G1 phase, and a positive-feedback loop regulating the start of the cell cycle in
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Tracking Cells and Monitoring Proliferation

2014
In many areas of immunology it is desirable to be able to track particular cells throughout an assay, whether it be in vivo or in vitro. There are two classes or reagents used for this purpose-general protein labels (reactive compounds that form random covalent bonds with amino groups on cellular proteins) and general membrane labels (lipophilic ...
Tosevski, Vinko, Mair, Florian
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Metabolism of Proliferating Cells

Cold Spring Harbor Perspectives in Biology, 2021
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Oncogenes and cell proliferation

Current Opinion in Genetics & Development, 1999
N, Dyson, A, Balmain
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