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Cell proliferation in neuroblastoma
European Journal of Cancer (1965), 1970Abstract In a patient with generalized neuroblastoma labeling index and median grain-count determinations on tumor cells in mitosis and interphase were performed autoradiographically after pulse-labeling with tritiated thymidine. The tumor-cell samples were removed from different parts of a superficially growing metastasis. The mitotic index was 0·57%
H P, Wagner, H, Käser
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Cancer, 1977
Much effort has gone into understanding the factors which regulate the growth of mammalian cells. Normal cells can exist in a proliferating state, or they can leave the cell division cycle and pass into a resting state, where they remain viable and functional for long periods.
A B, Pardee, R, Dubrow
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Much effort has gone into understanding the factors which regulate the growth of mammalian cells. Normal cells can exist in a proliferating state, or they can leave the cell division cycle and pass into a resting state, where they remain viable and functional for long periods.
A B, Pardee, R, Dubrow
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Oncogenes and cell proliferation
Current Opinion in Genetics & Development, 1995Abstract The control of cell proliferation has an impact on both basic and practical problems in biology. Many of these problems intersect in the study of cancer biology, which is one organizing focus of this issue of Current Opinion in Genetics & Development.
A J, Levine, J R, Broach
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CALCIUM AND CELL PROLIFERATION
British Medical Bulletin, 1986La premiere des reponses aux mitogenes est l'elevation du calcium intracellulaire avec une synthese d'inositol trisphosphate-1,4,5.
J C, Metcalfe +3 more
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Cell proliferation and apoptosis
Current Opinion in Cell Biology, 1999Cell proliferation and cell death are essential yet opposing cellular processes. Crosstalk between these processes promotes a balance between proliferation and death, and it limits the growth and survival of cells with oncogenic mutations. New insights into the mechanisms by which strong signals to proliferate and activation of cyclin-dependent kinases
Guo, Ming, Hay, Bruce A.
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The dynamics of cell proliferation
Medical Hypotheses, 2004The article provides a mathematical description based on the theory of differential equations, for the proliferation of malignant cells (cancer). A model is developed which enables us to describe and predict the dynamics of cell proliferation much better than by using ordinary curve fitting procedures.
John F, Moxnes +2 more
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Cell Proliferation in Carcinogenesis
Science, 1990Chemicals that induce cancer at high doses in animal bioassays often fail to fit the traditional characterization of genotoxins. Many of these nongenotoxic compounds (such as sodium saccharin) have in common the property that they increase cell proliferation in the target organ.
S M, Cohen, L B, Ellwein
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The Journal of Pathology, 1991
AbstractThe composition of a series of critical summaries is necessarily idiosyncratic, but in this compliation I have been guided by the recent publication of reviews and monographs on the subject.1–3 consequently, it seems reasonable to focus on more fundamental areas of research which may lie outside the normal reading of most pathologists ...
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AbstractThe composition of a series of critical summaries is necessarily idiosyncratic, but in this compliation I have been guided by the recent publication of reviews and monographs on the subject.1–3 consequently, it seems reasonable to focus on more fundamental areas of research which may lie outside the normal reading of most pathologists ...
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Regulation of cell proliferation and cell death by peroxisome proliferators
Microscopy Research and Technique, 2003AbstractPeroxisome proliferators cause increases in liver mass in rodents, linked to changes in cell proliferation and cell death of hepatocytes. These effects are reversible upon cessation of treatment. The underlying mechanism of the response in rodent liver is complex, but clearly dependent on activation of the nuclear receptor PPARα.
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