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Thrombin Stimulation of Renal Cells

Seminars in Thrombosis and Hemostasis, 1996
Numerous glomerular and vascular nephritides are associated with fibrin formation and deposition within the kidney. Thrombin, which induces fibrin formation, also exerts numerous cellular effects on both circulating cells and intrinsic glomerular cells through the activation of the functional thrombin receptor.
E, Rondeau   +3 more
openaire   +2 more sources

Contact stimulation of cell migration

Journal of Cell Science, 1992
ABSTRACT Mass migrations of dense cell populations occur periodically during embryonic development. It is known that extracellular matrices, through which the cells migrate, facilitate locomotion. However, this does not explain how cells, such as neural crest, can migrate as a dense cohort of cells in essentially continuous contact with ...
L A, Thomas, K M, Yamada
openaire   +2 more sources

Lactate stimulates endothelial cell migration

Wound Repair and Regeneration, 2006
ABSTRACTThe significance of the high lactate levels that characterize healing wounds is not fully understood. Lactate has been shown to enhance collagen synthesis by fibroblasts and vascular endothelial growth factor (VEGF) production by macrophages and endothelial cells. VEGF has been shown to induce endothelial cell migration.
Stefan, Beckert   +6 more
openaire   +2 more sources

Cell stimulation by haemopoietic lymphokines

Immunology Letters, 1987
Haemopoietic cells appear to respond to two distinct classes of growth factors, cell-bound molecules and lymphokines. Stimulation by lymphokines can be modelled by IL-3-dependent cell lines, and evidence is presented that such stimulation may represent a novel system of cell signalling unrelated to those where stimulation is a progression from a ...
openaire   +3 more sources

Stimulation of Cells by Antibody

Science, 1973
Tumor cell lines exposed to immunoglobulins specific for cell surface antigens developed increased cellular incorporation of [ 125 I]iododeoxyuridine and [ 3 H]thymidine (up to 200-fold increases over cells treated with normal rabbit immunoglobulins). Antibody-stimulated cells multiplied more
W T, Shearer, G W, Philpott, C W, Parker
openaire   +2 more sources

Galectin-3 Stimulates Cell Proliferation

Experimental Cell Research, 1998
Galectin-3, a beta-galactoside-binding protein, has been shown to be involved in multiple biological processes through interaction with its complementary glycoconjugates. Here we provide the first evidence of galectin-3 as a mitogen. Incubation of quiescent cultures of normal human lung fibroblast IMR-90 cells with recombinant galectin-3 (rgalectin-3 ...
H, Inohara, S, Akahani, A, Raz
openaire   +2 more sources

Electromagnetic Cell Stimulation

1996
Electromagnetic field (ELF) stimulation of biological tissues has been, and remains, a contentious issue in the scientific world. Many of the experiments have not been reproducible in other laboratories, and many appear not to have been thoroughly conducted.
D. Jones, B. McLeod
openaire   +1 more source

Ovarian Stimulation and Granulosa-Cell Tumour

Obstetrical & Gynecological Survey, 1993
Ovarian stimulation in the treatment of infertility is far from physiological because patients and their ovaries are exposed to high concentrations of gonadotropins. Many studies have focused on the two most common side-effects of ovarian stimulation--ie, hyperstimulation and multiple pregnancy. We describe 12 patients in whom granulosa-cell tumour was
W, Willemsen   +4 more
openaire   +2 more sources

Stimulation of haemopoietic stem cell proliferation: Characteristics of the stimulator-producing cells

Leukemia Research, 1982
Media conditioned by regenerating murine bone marrow cells contain a stimulator of haemopoietic stem cell proliferation. Fractionated cell populations have been examined for production of this stimulatory activity in order to characterize its cellular source.
E G, Wright   +3 more
openaire   +2 more sources

Senescence Stimulates U937-Endothelial Cell Interactions

Experimental Cell Research, 1993
Progressive pathophysiologic modifications of endothelial cells are associated with aging. In vitro, endothelial cell senescence is accompanied by the failure to proliferate as well as by perturbations in gene expression. Here we show that (i) senescence enhances monoblastoid U937 cell adhesion to the endothelial monolayer; (ii) the enhanced ...
J. A. Maier, M. Statuto, G. Ragnotti
openaire   +3 more sources

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