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The Effects of Photobiomodulation Therapy on Tensile-Cultured Cementoblasts Cells

Photobiomodulation, photomedicine, and laser surgery
Background: Studies show that photobiomodulation therapy (PBMT) boosts cellular ATP production and cell growth and reduces inflammation. Additionally, mechanical tension affects gene expression, impacting cellular functions like proliferation and ...
Chuan-Yi Kao   +3 more
semanticscholar   +1 more source

Comparative effects of parathyroid hormone on osteoblasts and cementoblasts

Journal of Clinical Periodontology, 1987
Abstract Although bone, dentin and dental cementum are mesenchymal mineralized tissues composed mainly of collagen and hydroxy apatite, they differ markedly in their suceptibility to resorption. Bone undergoes physiological resorption to which the dental tissues appear to be resistant.
S, Lindskog   +2 more
openaire   +2 more sources

IL1β inhibits differentiation of cementoblasts via microRNA‐325‐3p

Journal of Cellular Biochemistry, 2019
AbstractCementum regeneration is considered the gold standard for the treatment of periodontitis. As one of the most important primary proinflammatory cytokines, interleukin 1β (IL1β) plays an essential role during the early stage of periodontitis and its amounts simultaneously increase dramatically during this stage.
Yuzhuo Wang   +5 more
openaire   +2 more sources

PGE2Activates Cementoclastogenesis by CementoblastsviaEP4

Journal of Dental Research, 2007
Destruction of cementum and alveolar bone is the main causative event for the exfoliation of teeth as a consequence of periodontitis. Prostaglandin E2(PGE2) and PGE receptor subtypes (EPs) play an important role in modulating osteoblast-mediated osteoclastogenesis; however, no information is available on the role of PGE2and EPs in regulating ...
H, Oka   +7 more
openaire   +2 more sources

Inhibition of AXUD1 attenuates compression-dependent apoptosis of cementoblasts

Clinical Oral Investigations, 2016
Root resorptions are common undesirable side effects of orthodontic treatment. In most patients, these defects are repaired by cementoblasts. However, in 1-5 % of patients, the repair fails. The repair mechanism is not well understood. Apoptosis of cementoblasts might contribute to an impaired repair of root resorptions induced by orthodontic forces.To
Katja, Korb   +6 more
openaire   +2 more sources

MicroRNA-155-3p Mediates TNF-α-Inhibited Cementoblast Differentiation

Journal of Dental Research, 2017
Periodontitis is a prevalent and chronic inflammatory disease that is interrelated with systemic health. Periodontitis can be promoted by tumor necrosis factor α (TNF-α). Cementum, a vital part of the periodontium, is a bone-like mineralized tissue that is produced by cementoblasts.
X, Wang   +6 more
openaire   +2 more sources

Sodium fluoride causes oxidative stress and apoptosis in cementoblasts

Chemico-Biological Interactions, 2018
Toothpaste with fluoride concentration up to 5000 ppm are recommended to the patients who are susceptible to root caries; however, the effects of fluoride on cementoblasts have received less attention.The OCCM-30 cells were exposed to 0,0.5, 5, 10 mM NaF respectively. A TUNEL (TdT-mediated dUTP-biotin nick end labeling) assay kit was used to detect the
Jing, Ni   +4 more
openaire   +2 more sources

Effects of TNF-α on Cementoblast Differentiation, Mineralization, and Apoptosis

Journal of Dental Research, 2015
Tumor necrosis factor–α (TNF-α) is involved in various inflammatory processes, including periodontitis. Although the influences of TNF-α on periodontal ligament fibroblasts and osteoblasts have been widely documented, its effects on cementoblasts, the cells responsible for cementum production, remain largely unknown. In this study, we found that TNF-α
Y L, Wang   +9 more
openaire   +2 more sources

Prostanoid– and Interleukin‐1–Induced Primary Genes in Cementoblastic Cells

Journal of Periodontology, 2006
Background: Cementum is a key component of a functional periodontal organ. However, regenerating lost cementum is difficult and often incomplete. Identifying molecular mediators of cementoblast differentiation and function should lead to better targeted treatment for periodontitis.
Jeanne M, Nervina   +3 more
openaire   +2 more sources

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