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Transcranial Doppler in the Diagnosis of Cerebral Vasospasm: An Updated Meta-Analysis
Critical Care Medicine, 2018Objectives: To evaluate the performance of transcranial Doppler and transcranial color-coded duplex Doppler in patients with cerebral vasospasm due to aneurysm rupture. Angiography was considered as the gold standard comparator.
Jean-Mathieu Mastantuono +4 more
semanticscholar +1 more source
Neurosurgery, 2019
BACKGROUND Whether delayed cerebral infarction (DCIn) after aneurysmal subarachnoid hemorrhage (aSAH) is driven by large artery vasospasm is still controversial.
Jonathan Brami +9 more
semanticscholar +1 more source
BACKGROUND Whether delayed cerebral infarction (DCIn) after aneurysmal subarachnoid hemorrhage (aSAH) is driven by large artery vasospasm is still controversial.
Jonathan Brami +9 more
semanticscholar +1 more source
Neurosurgery Quarterly, 1995
Publisher Summary This chapter focuses on cerebral vasospasm. Vasospasm is the delayed onset of focal or diffuse narrowing of the major cerebral arteries days after subarachnoid hemorrhage (SAH). Vasospasm affects the intradural arteries in the subarachnoid space. Subarachnoid bleeding may result from trauma, ruptured aneurysms, vascular malformations
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Publisher Summary This chapter focuses on cerebral vasospasm. Vasospasm is the delayed onset of focal or diffuse narrowing of the major cerebral arteries days after subarachnoid hemorrhage (SAH). Vasospasm affects the intradural arteries in the subarachnoid space. Subarachnoid bleeding may result from trauma, ruptured aneurysms, vascular malformations
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Critical Care Medicine, 2018
Objectives: To investigate the release of proinflammatory damage-associated molecular pattern molecule “high-mobility group box-1” in the serum of patients after aneurysmal subarachnoid hemorrhage and its association with cerebral vasospasm.
S. R. Chaudhry +9 more
semanticscholar +1 more source
Objectives: To investigate the release of proinflammatory damage-associated molecular pattern molecule “high-mobility group box-1” in the serum of patients after aneurysmal subarachnoid hemorrhage and its association with cerebral vasospasm.
S. R. Chaudhry +9 more
semanticscholar +1 more source
Contemporary Neurosurgery, 1988
Cerebral vasospasm (specifically, intracranial arterial spasm) is variously defined as: (1) an arteriographically evident narrowing of the lumen of one or more of the major intracranial arteries at the base of the brain due to contraction of the smooth muscle within the arterial wall, or due to the morphological changes in the arterial wall and along ...
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Cerebral vasospasm (specifically, intracranial arterial spasm) is variously defined as: (1) an arteriographically evident narrowing of the lumen of one or more of the major intracranial arteries at the base of the brain due to contraction of the smooth muscle within the arterial wall, or due to the morphological changes in the arterial wall and along ...
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Pathogenesis of Cerebral Vasospasm
Neurological Research, 1980Cerebral arteries have been shown to react to experimental subarachnoid hemorrhage with a nonspecific arterial injury reaction characterized by endothelial cell desquamation, adherence of platelets to the exposed collagen, subendothelial edema, and medial necrosis.
Branson Pj, Alksne Jf
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Hypervolemia in Cerebral Vasospasm [PDF]
Journal of Neurosurgery, 2006 Object. Arterial vasospasm is the most common cause of delayed ischemic neurological deficits (DINDs) and one of the major causes of disability following subarachnoid hemorrhage (SAH). Current management of vasospasm involves intravascular volume expansion and hemodynamic augmentation with the goal of increasing cerebral blood flow (CBF).
Bertil Romner, Andreas Raabe
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Neurosurgery Clinics of North America, 1998
Established vasospasm is refractory to vasodilators, although certain agents (nimodipine or papavarine) may reverse early vasospasm when administered in high local concentrations. Calcium channel antagonists do not affect the incidence of arteriographic vasospasm and probably improve outcome by other mechanisms.
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Established vasospasm is refractory to vasodilators, although certain agents (nimodipine or papavarine) may reverse early vasospasm when administered in high local concentrations. Calcium channel antagonists do not affect the incidence of arteriographic vasospasm and probably improve outcome by other mechanisms.
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Etiology of Cerebral Vasospasm
1999Cerebral vasospasm is a gradual onset and prolonged constriction of the cerebral arteries in the subarachnoid space after subarachnoid hemorrhage. The principal cause is the surrounding blood clot. The significance of vasospasm is that flow through the constricted arteries may be reduced sufficiently to cause cerebral infarction.
Marcus A. Stoodley +2 more
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Cerebrovascular Diseases, 2016
Background: Several clinical studies have indicated the efficacy of cilostazol, a selective inhibitor of phosphodiesterase 3, in preventing cerebral vasospasm after aneurysmal subarachnoid hemorrhage (SAH). They were not double-blinded trial resulting in
Naoya Matsuda +7 more
semanticscholar +1 more source
Background: Several clinical studies have indicated the efficacy of cilostazol, a selective inhibitor of phosphodiesterase 3, in preventing cerebral vasospasm after aneurysmal subarachnoid hemorrhage (SAH). They were not double-blinded trial resulting in
Naoya Matsuda +7 more
semanticscholar +1 more source