Results 191 to 200 of about 91,813 (306)
On why cancer cells require a great amount of glucose
Abstract The traditional thinking has been that cancer cells require a great amount of glucose to support their rapid growth, but the reality may be different. We have previously demonstrated that all cancer cells in The Cancer Genome Atlas harbor persistent Fenton reactions in their cytosol, which generate OH− ${\text{OH}}^{-}$ and ultimately kill the
Xuechen Mu +6 more
wiley +1 more source
Identification and experimental validation of copper metabolism-related genes in cervical cancer. [PDF]
Ye Y, Wang Y, Gong D, Zhao Y, Wei X.
europepmc +1 more source
Fractures of lower cervical vertebrae in the presence of a congenital cleft of the atlas
Gade, K.S.R. +3 more
openaire +1 more source
Background and Purpose EZH2 (enhancer of zeste homologue 2) inhibitors are an emerging class of drugs that target epigenetic regulation. However, their efficacy in solid tumours has been limited, partly due to drug‐induced upregulation of fatty acid synthesis.
Rui Cheng +10 more
wiley +1 more source
Resection and 3D-printed prosthetic reconstruction of a giant aneurysmal bone cyst in the atlas: illustrative case. [PDF]
Zheng Y +7 more
europepmc +1 more source
Anatomy of the woodchuck (Marmota monax) / [PDF]
Bezuidenhout, A. J. (Abraham Johannes), 1942- +1 more
core +2 more sources
ABSTRACT Aim To combine qualitative and quantitative data to evaluate the feasibility, participant satisfaction and effectiveness of a hybrid pulmonary rehabilitation programme following hospital discharge for an exacerbation of chronic obstructive pulmonary disease (COPD).
Sarah Gephine +4 more
wiley +1 more source
A Case of Initial Missed Diagnosis of Fatal Posterior Atlantoaxial Dislocation Without Odontoid Fracture. [PDF]
Hagino T, Hagino T, Ohba T.
europepmc +1 more source
Characteristics of The Cancer Genome Atlas cases relative to U.S. general population cancer cases [PDF]
Bailey, Matthew H +5 more
core +1 more source
Correlation of the differential expression of PIK3R1 and its spliced variant, p55α, in pan‐cancer
PIK3R1 undergoes alternative splicing to generate the isoforms, p85α and p55α. By combining large patient datasets with laboratory experiments, we show that PIK3R1 spliced variants shape cancer behavior. While tumors lose the protective p85α isoform, p55α is overexpressed, changes linked to poorer survival and more pronounced in African American ...
Ishita Gupta +10 more
wiley +1 more source

