Vitamin D supplementation in individuals with type 1 diabetes mellitus (T1DM): a systematic review and meta-analysis focusing on pediatrics. [PDF]
Fan S, Zhang T, Yin L.
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Best Practice Recommendations for the Assessment, Prevention and Treatment of Vitamin D Deficiency in Türkiye: A 2026 Update in a Setting with Limited Mandatory Food Fortification. [PDF]
Gogas Yavuz D +9 more
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Vitamin D and Hemopoietic Stem Cell Transplantation: Clinical Guidance for GVHD Management and Post-Transplant Outcomes. [PDF]
Fazio M +6 more
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Higher prevalence of vitamin D deficiency among arthroplasty patients presenting in winter. [PDF]
Beadel H, Sandiford NA.
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Erratum to "Treatment of Hypovitaminosis D With Cholecalciferol in Dogs With Protein-Losing Enteropathies: A Randomized, Double-Blind, Placebo-Controlled, Clinical Trial". [PDF]
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The primary source of exposure to cholecalciferol in dogs and cats is ingestion of rodenticide baits with vitamin D3 as the active ingredient. Other sources of this toxin are human medications and rarely, contaminated pet food. Although the reported lethal dose 50% for cholecalciferol is 88 mg/kg, deaths have been seen with an individual exposure of 2 ...
Michael E, Peterson, Kerstin, Fluegeman
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Ergocalciferol and Cholecalciferol in CKD
American Journal of Kidney Diseases, 2012The development of chronic kidney disease (CKD) is accompanied by a progressive decrease in the ability to produce 1,25-dihydroxyvitamin D. Pharmacological replacement with active vitamin D therefore has been a cornerstone of secondary hyperparathyroidism therapy in the end-stage renal disease population treated by long-term dialysis.
Sagar U, Nigwekar +2 more
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Cholecalciferol rodenticide intoxication in a cat
Journal of the American Veterinary Medical Association, 1991A 4-month-old 2.5-kg sexually intact female domestic shorthair cat was referred to the teaching hospital because of suspected cholecalciferol intoxication after ingestion of a cholecalciferol-containing rodenticide. At referral, the cat was hypercalcemic, hyperkalemic, and acidotic. Despite management of hypercalcemia and preservation of renal function
E N, Peterson +3 more
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The selenium dioxide oxidation of cholecalciferol
Steroids, 1977Abstract The selenium oxidation of cholecalciferol afforded a mixture of products from which la- and lβ-hydroxycholecalciferols were isolated.
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Fecal Loss of Cholecalciferol in Gastrectomized Rats
Digestion, 1986Rats were gastrectomized, and the intestinal absorption and fecal excretion of cholecalciferol were studied following the administration of radioactive cholecalciferol, either by subcutaneous injection or with the aid of a gastric tube. From measurements of radioactivity in feces and sera it has been possible to establish that gastrectomy in rats ...
M S, Meyer +4 more
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