Results 71 to 80 of about 13,261 (250)

Sensory Nerve Conduction Amplitudes and Z‐Compounds Depend on Body Mass Index in Overweight and Obese Individuals

open access: yesMuscle &Nerve, Volume 71, Issue 6, Page 1052-1062, June 2025.
ABSTRACT Introduction/Aims The association between body mass index (BMI) and nerve conduction study (NCS) amplitudes has not been adequately studied in overweight and obese subjects. Our aim was to estimate the impact of high BMI on NCS amplitudes and NCS summary measures.
Trond Sand   +6 more
wiley   +1 more source

Mechanisms of IVIG Efficacy in Chronic Inflammatory Demyelinating Polyneuropathy [PDF]

open access: yes, 2018
Background: Chronic inflammatory demyelinating polyneuropathy (CIDP) is the most common treatable acquired chronic polyneuropathy. Corticosteroids, plasmapheresis and intravenous immunoglobulins (IVIG) have been shown to be effective in randomized ...
Lünemann, Jan   +2 more
core  

Clinical applications of immunoglobulin in neuromuscular diseases: focus on inflammatory myopathies [PDF]

open access: yes, 2014
During recent years, an increasing number of neuromuscular diseases have been recognized either to be caused primarily by autoimmune mechanisms, or to have important autoimmune components.
Oliveira, Acary Souza Bulle   +2 more
core   +3 more sources

Nerve Diameter and DTI Parameters Maybe Potential Markers for Clinical Trial in Patients With Charcot–Marie–Tooth Disease Type 1A

open access: yesEuropean Journal of Neurology, Volume 32, Issue 6, June 2025.
ABSTRACT Background Charcot–Marie–Tooth disease type 1A (CMT1A) is the most common form of CMT, varying from asymptomatic to severe. The aim of this study was to investigate whether the diffusion tensor imaging (DTI) parameters and diameter of the proximal nerves, as measured by magnetic resonance neurography, can be used as a potential marker to ...
Yuchen Zhu   +3 more
wiley   +1 more source

Pitfalls in the diagnostic evaluation of subacute combined degeneration [PDF]

open access: yes, 2015
We report a case of a 43-year-old man presenting with a 2-week history of painless ascending sensory disturbances, suspected to be suffering from acute inflammatory polyneuropathy.
Linnebank, M   +3 more
core   +1 more source

Clinical and Electrophysiological Characterization of Diabetic Neuropathy in a Sub‐Saharan African Cohort

open access: yesJournal of the Peripheral Nervous System, Volume 30, Issue 2, June 2025.
ABSTRACT Background Diabetic neuropathy (DN) is the most frequent complication of diabetes mellitus, contributing to increased morbidity and mortality. Previous clinical studies on DN in sub‐Saharan Africa (sSA) have used purely clinical approaches, potentially underestimating the true magnitude of this disease. This study was designed to determine the
Samuel Eric Chokote   +16 more
wiley   +1 more source

Neurofascin antibodies in autoimmune, genetic, and idiopathic neuropathies [PDF]

open access: yes, 2017
OBJECTIVE: To measure the frequency, persistence, isoform specificity, and clinical correlates of neurofascin antibodies in patients with peripheral neuropathies.
Burnor, E   +8 more
core   +1 more source

Serum neurofilament light chain in chronic inflammatory demyelinating polyneuropathy

open access: yesBrain and Behavior, 2021
Objectives Neurofilament light chain (NfL) levels have been suggested as reflecting axonal damage in various inflammatory and neurodegenerative disorders, including acquired peripheral neuropathies.
Tomohiro Hayashi   +9 more
doaj   +1 more source

Albuminocytological dissociation in different electrophysiological gbs variants [PDF]

open access: yes, 2015
The objective of our study was to determine the distribution of different electrophysiological variants of GBS and its relationship with albuminocytological dissociation (ACD).
Badshah, Mazhar   +6 more
core   +1 more source

A patient with amyotrophic lateral sclerosis and atypical clinical and electrodiagnostic features: a case report [PDF]

open access: yes, 2011
INTRODUCTION: Amyotrophic lateral sclerosis is a rapidly progressive, fatal neurodegenerative disorder for which there is no effective treatment. The diagnosis is dependent on the clinical presentation and consistent electrodiagnostic studies. Typically,
Alexander Venizelos   +2 more
core   +2 more sources

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