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Abstract Objective SCN2A pathogenic mutations, such as the recurrent heterozygous Nav1.2‐L1342P, are monogenic causes of epilepsy. In this human‐induced pluripotent stem cell–derived model system, we aim to investigate the molecular and cellular mechanisms underlying SCN2A‐L1342P‐associated pathology. Methods Using a human male induced pluripotent stem
Maria I. Olivero‐Acosta +26 more
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Digital distraction and adolescent development: a narrative review of smartphone use, cognitive effects, and policy interventions. [PDF]
Colakoglu D, van Golen C.
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Overview of Colistin Resistance in the Middle East and North Africa (MENA) Region. [PDF]
Saniour R +5 more
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Impact of the Arrangement Modes of the Antimicrobial Motifs WWR-7 and its Analogs in CAMPs on Their Therapeutic Properties. [PDF]
Yadav M +4 more
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Self-spoliation and forms of resistance in total institutions: an exploration of time and space in an Albanian communist regime internment camp. [PDF]
Floridi F +3 more
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Seasonal Climate Drivers and Immunohematological Profiles of Malaria in Rohingya Refugee Camps, Bangladesh: A 4-Year Repeated Cross-Sectional Study. [PDF]
Islam A +10 more
europepmc +1 more source
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The Concentration Camp Syndrome
Australian & New Zealand Journal of Psychiatry, 1994A psychiatric syndrome following overwhelming stress after an interval of more than thirty years is described in holocaust survivors who had claimed compensation for persecution between 1939 and 1945. Five nuclear symptom complexes emerge: depressive reactions; anxiety states; somatic complaints; subjective intellectual impairment ...
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Abstract This book explains why concentration camps were created, and how they changed radically in the course of the twentieth century to become instruments of mass terror and genocide. In popular perception, concentration camps are synonymous with genocide—racial extermination. Yet the great majority of them were not sites of genocide.
Rolf J Kleber, Danny Brom
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Rolf J Kleber, Danny Brom
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