Results 151 to 160 of about 516,098 (208)
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Update on coronary artery spasm 2022 – A narrative review
International Journal of Cardiology, 2022IMPORTANCE In today's era of precision medicine a renaissance of functional coronary disease including coronary artery spasm (CAS) can be observed. This development is fueled by novel diagnostic methods as well as first evidence from randomized trials ...
Andreas Seitz, Peter Ong, Udo Sechtem
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Mechanisms of Coronary Artery Spasm [PDF]
Circulation, 2011 The term coronary artery spasm (CAS) refers to a sudden, intense vasoconstriction of an epicardial coronary artery that causes vessel occlusion or near occlusion. Although CAS may be involved in other coronary syndromes, it represents the usual cause of variant angina.
Gaetano A Lanza, Giulia Careri
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New England Journal of Medicine, 1978
Detailed formal protocol with illustrations and extensive bibliography. Note: Some information has been redacted in the publicly-available version due to privacy issues. For questions, contact archives@utsouthwestern.edu.
Eugene Braunwald +2 more
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Detailed formal protocol with illustrations and extensive bibliography. Note: Some information has been redacted in the publicly-available version due to privacy issues. For questions, contact archives@utsouthwestern.edu.
Eugene Braunwald +2 more
exaly +3 more sources
Diseases of the Chest, 1968
SUMMARY In a series of 750 coronary cinearteriograms, coronary spasm was observed in only seven individuals. At the time of the cinearteriogram, three experienced an attack of chest pain that had all the features of coronary insufficiency before the spasm was relieved by nitroglycerin.
M A, Demany, A, Tambe, H A, Zimmerman
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SUMMARY In a series of 750 coronary cinearteriograms, coronary spasm was observed in only seven individuals. At the time of the cinearteriogram, three experienced an attack of chest pain that had all the features of coronary insufficiency before the spasm was relieved by nitroglycerin.
M A, Demany, A, Tambe, H A, Zimmerman
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Current Treatment Options in Cardiovascular Medicine, 2000
Attacks of variant angina usually respond promptly to sublingual administration of short-acting nitrates (nitroglycerin, 0.3 to 0.4 mg, or isosorbide dinitrate, 5 to 10 mg), which may be repeated after 3 to 5 minutes if pain persists. In the rare cases resistant to sublingual nitrates, sublingual nifedipine (5 to 10 mg) or, when readily available ...
, Lanza, , Maseri
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Attacks of variant angina usually respond promptly to sublingual administration of short-acting nitrates (nitroglycerin, 0.3 to 0.4 mg, or isosorbide dinitrate, 5 to 10 mg), which may be repeated after 3 to 5 minutes if pain persists. In the rare cases resistant to sublingual nitrates, sublingual nifedipine (5 to 10 mg) or, when readily available ...
, Lanza, , Maseri
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OESOPHAGEAL SPASM IN PATIENTS WITH CORONARY ARTERY SPASM
The Lancet, 1986Oesophageal manometry was performed in 20 patients with chest pain, of whom 10 showed coronary artery spasm (group I) and 10 did not (group II). In the basal state oesophageal spasms were recorded in 6 patients from group I but in none from group II (p less than 0.05).
Rasmussen, K +3 more
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JAMA: The Journal of the American Medical Association, 1976
Rarely has an etiologic concept bounced back so vigorously as that of coronary artery spasm. Only recently dismissed as a nonexistent entity—let alone as the cause of angina—it is now being implicated with increasing frequency in some cases, possibly types, of the anginal syndrome.
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Rarely has an etiologic concept bounced back so vigorously as that of coronary artery spasm. Only recently dismissed as a nonexistent entity—let alone as the cause of angina—it is now being implicated with increasing frequency in some cases, possibly types, of the anginal syndrome.
openaire +2 more sources
Stenting for coronary artery spasm
Catheterization and Cardiovascular Interventions, 2002AbstractWe evaluated coronary stenting in nine patients with clinically severe, angiographically documented spasm refractory to aggressive pharmacologic management. No patient subsequently developed unstable ischemia requiring hospitalization as a consequence of recurrent spasm within the stent. Mechanisms of therapeutic failure included both peristent
Shailesh, Khatri +3 more
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