Results 221 to 230 of about 11,009,796 (379)

Explaining the decrease in U.S. deaths from coronary disease, 1980-2000.

open access: yesNew England Journal of Medicine, 2007
E. Ford   +7 more
semanticscholar   +1 more source

Alpinetin Nanoparticles Alleviate Optic Nerve Injury Induced by Acute Glaucoma via LRP1‐PPARγ Mediated Regulation of Microglial Lipid Metabolism

open access: yesAdvanced Science, EarlyView.
Microglial lipid metabolic dysfunction drives neurodegeneration in glaucoma. We found loss of LRP1 causes lipid accumulation and inflammation. We developed alpinetin‐loaded nanoparticles (AlpNPs) that bind LRP1, activate the PPARγ‐LXRα‐ABCA1 pathway to restore lipid homeostasis, promote an anti‐inflammatory phenotype, and protect retinal ganglion cells,
Miao Wei   +8 more
wiley   +1 more source

Corrigendum to “The “L-Sandwich” Strategy for True Coronary Bifurcation Lesions: A Randomized Clinical Trial”

open access: yesJournal of Interventional Cardiology
Quan Guo   +7 more
doaj   +1 more source

Six-Year Outcomes of CABG vs PCI in Diabetic Patients with Multivessel Coronary Disease. [PDF]

open access: yesMed Sci Monit
Jonik S   +6 more
europepmc   +1 more source

Research on Zheng Classification Fusing Pulse Parameters in Coronary Heart Disease [PDF]

open access: hybrid, 2013
Rui Guo   +7 more
openalex   +1 more source

[2017 ESC focused update on dual antiplatelet therapy in coronary artery disease developed in collaboration with EACTS.]

open access: yesKardiologia polska, 2018
M. Valgimigli   +16 more
semanticscholar   +1 more source

Surface‐Associated Proteins on Extracellular Vesicles Remodel the Tumor Microenvironment by Potentiating TGF‐β Signaling in a Contact‐Dependent Manner

open access: yesAdvanced Science, EarlyView.
Extracellular vesicles (EVs) released from TGF‐β‐activated CAFs are enriched with ECM proteins such as TSG6 and THBS1, which facilitate their binding to recipient cell membranes. This EV–cell interaction promotes the clustering of CD44 and TGF‐β receptors on the target cell surface, thereby potentiating TGF‐β signaling activity. This study highlights a
Chao Li   +7 more
wiley   +1 more source

Self‐Trapped Excitons in Carbon Quantum Dots with Large NIR‐II Photo‐Thermoelectric Catalysis Induce Pyroptosis for Cancer Therapy

open access: yesAdvanced Science, EarlyView.
The self‐trapped excitons in carbon quantum dots can serve as NIR‐II absorbers, which efficiently induce pyroptosis by driving the generation of reactive oxygen species through mild NIR‐II photothermal effect. Combined with local photophysical therapy and the cascade reaction of pyroptosis, it can activate systemic immunity and prevent tumor recurrence.
Tesen Zhang   +10 more
wiley   +1 more source

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