Results 221 to 230 of about 40,540 (250)
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Immunomodulation by adoptive regulatory T‐cell transfer improves Coxsackievirus B3‐induced myocarditis

The FASEB Journal, 2018
Regulatory T (Treg) cells offer new therapeutic options for controlling undesired systemic and local immune responses. The aim of the current study was to determine the impact of therapeutic Treg administration on systemic and cardiac inflammation and ...
K. Pappritz   +16 more
semanticscholar   +1 more source

COXSACKIEVIRUS B3 IN HUMAN MILK

The Pediatric Infectious Disease Journal, 2006
Coxsackievirus B3 can cause severe neonatal disease with high mortality. We present the first report of detection of coxsackievirus B3 in the mothers' milk of 2 severely infected neonates by culture and reverse transcriptase-polymerase chain reaction. Reverse transcriptase-polymerase chain reaction is a rapid and sensitive tool to detect coxsackievirus
Mei-Ling, Chang   +5 more
openaire   +2 more sources

Group B Coxsackievirus Virulence

2008
That which is understood of virulence phenotypes in the picornaviruses derives in large part from studies of artificially attenuating phenotypes rather than through examination of naturally occurring virus strains. The CVB replicate well in a variety of different murine and human cell cultures, making them excellent viruses with which to engage the ...
S, Tracy, C, Gauntt
openaire   +2 more sources

Genetics of coxsackievirus B3 cardiovirulence

European Heart Journal, 1995
The human enteroviruses, especially the coxsackie B viruses, have been established as aetiologic agents of human inflammatory heart disease, a condition which may lead to dilated cardiomyopathy and heart failure. It is clear from murine models of coxsackievirus B3-induced inflammatory heart disease that not all strains of the virus are cardiovirulent ...
S, Tracy, Z, Tu, N, Chapman, G, Hufnagel
openaire   +2 more sources

Coxsackievirus and Congenital Malformation

JAMA: The Journal of the American Medical Association, 1967
To the Editor:— I have read with particular interest the communication by Brown and Evans, "Serologic Evidence of Coxsackievirus Etiology of Congenital Heart Disease" (199: 183, 1967), because we have found a possible relation between the infections of coxsackievirus B during pregnancy and fetal deaths.
openaire   +2 more sources

Replicase gene of coxsackievirus B3

Journal of Virology, 1984
A cDNA copy covering two-thirds of the coxsackievirus B3 genome was cloned in the PstI site of the pBR322 vector. A nucleotide sequence containing the gene for the viral replicase and the 3' noncoding region of the coxsackievirus B3 genome was determined.
P O, Stålhandske   +2 more
openaire   +2 more sources

Coxsackievirus in an Infant Chimpanzee

Journal of Medical Primatology, 1978
Coxsackie B viruses may cause a severe, often fatal, illness in newborn and infant human subjects. As recorded in this case, infant chimpanzees respond similarly to Coxsackie B-5 virus.
M E, Kelly   +3 more
openaire   +2 more sources

Enteric resistance and coxsackievirus B

The American Journal of Clinical Nutrition, 1977
Sidney Kibrick,4 M.D., Ph.D. and Roger M. Loria, Ph.D.Although the oro- and nasopharynx serveas portals of entry for many different vi-nuses, the number of these agents that reachthe gut and initiate primary infection at thissite is limited. Many swallowed viruses areprobably inactivated by the acidic and pro-teolytic secretions of the stomach that ...
S, Kibrick, R M, Loria
openaire   +2 more sources

Autoimmunity in Coxsackievirus Infection

2008
Abstract Viral infections frequently result in the production of autoantibodies. In most cases, these autoantibodies are low-affinity IgMs that exhibit extensive cross-reactions. Sometimes these virus-triggered immune responses progress to a pathogenic autoimmunity to form autoimmune disease.
openaire   +2 more sources

The Coxsackievirus and Adenovirus Receptor

2008
The coxsackievirus and adenovirus receptor (CAR) has been studied extensively since its identification and isolation in 1997. The CAR is an immunoglobulin superfamily protein with two extracellular Ig-like domains, a single membrane-spanning sequence, and a significant cytoplasmic domain.
P, Freimuth, L, Philipson, S D, Carson
openaire   +2 more sources

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