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Multiple cranial nerve palsies secondary to a recurrence of Hansen's disease. [PDF]
BMJ Case Rep, 2018 Shah PM, Dhakre VW, Prasad A, Samdani P.
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Slowly progressive cranial nerve palsies
Medical Journal of Australia, 2006Perineural spread (PNS) from these lesionsinvolves either small nerves, identified at pathological examina-tion (incidental), or large nerves, presenting clinically as cranialnerve palsies. Basal cell carcinoma is the more common skincancer, but incidental PNS is most frequently associated withsquamous cell carcinoma.
Warner, Giles C. +2 more
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2013
This chapter describes the neuroanatomy and common lower cranial neuropathies, including clinical presentation, evaluation, diagnosis, and treatment of the following cranial nerves: trigeminal (fifth), facial (seventh), glossopharyngeal (ninth), vagus (tenth), spinal accessory (eleventh), and hypoglossal (twelfth).
Vita Grynova Kesner, Christina Fournier
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This chapter describes the neuroanatomy and common lower cranial neuropathies, including clinical presentation, evaluation, diagnosis, and treatment of the following cranial nerves: trigeminal (fifth), facial (seventh), glossopharyngeal (ninth), vagus (tenth), spinal accessory (eleventh), and hypoglossal (twelfth).
Vita Grynova Kesner, Christina Fournier
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Sensory ataxia with cranial nerve palsies
Practical Neurology, 2021A previously independent 64-year-old man presented to his local hospital with new-onset diplopia and dysarthria while watching television. Over the following days, he developed painless perioral and upper limb sensory disturbance, gait unsteadiness and limb ataxia, most pronounced in the upper limbs.
Alexander, Grundmann +3 more
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2015
The clinical features of superior oblique paresis vary widely depending on the type. Common features include an ipsilateral hypertropia that increases on contralateral gaze, and a positive head tilt test with the hypertropia increasing on head tilt to the side of the hypertropia.
Kenneth W. Wright, Yi Ning J. Strube
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The clinical features of superior oblique paresis vary widely depending on the type. Common features include an ipsilateral hypertropia that increases on contralateral gaze, and a positive head tilt test with the hypertropia increasing on head tilt to the side of the hypertropia.
Kenneth W. Wright, Yi Ning J. Strube
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Pediatric Cranial Nerve Palsies
Journal of Pediatric Neurology, 2016The diagnostic evaluation and treatment of children with cranial nerve palsies is markedly different from that of adults. Genetic, traumatic, inflammatory, and compressive etiologies are common. Unlike adults, vascular etiologies are rare. Treatment entails addressing factors such as amblyopia and fusion.
Paul Phillips, Brita Rook
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Familial recurrent cranial nerve palsies
Acta Neurologica Scandinavica, 1988Family cases of recurrent cranial nerve palsies are seldom reported. This paper presents a family with recurrent facial and ocular nerve palsies in 2 brothers. Their father and his sister had Bell's palsies. Examinations provided no explanation. Six previous reports of families with recurrent cranial nerve palsies are summarized. The pedigrees speak in
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