Results 141 to 150 of about 3,999,813 (301)

Valuing Australia's Creative Industries - Final Report - December 2013.pdf

open access: yes, 2015
Reports on the value of Australia's Creative Industries, giving an overview of the current sector, best practice, and the Nesta method. Profiles the Australian sector, with specific information on the creative industries workforce, employment, embedded ...
Creative Industries Innovation Centre, SGS Economics
core  

Differential expression of cancer‐related genes supports prediction of poor response to first‐line treatments in T‐ALL pediatric patients with high minimal residual disease

open access: yesMolecular Oncology, EarlyView.
In the present work, we have identified a transcriptional signature based on the differential expression of six genes (BCL2&MAST4, HSH2D&LAT2, METRN&PITPNM2) that would facilitate the early detection of T‐cell acute lymphoblastic leukemia (T‐ALL) patients prone to a poor treatment response and could be implemented at diagnosis, along with other risk ...
Antonio Lahera   +11 more
wiley   +1 more source

Penggunaan model pembelajaran creative problem solving untuk meningkatkan kemampuan berpikir kreatif dan motivasi belajar matematika siswa

open access: yesJP3M (Jurnal Penelitian Pendidikan dan Pengajaran Matematika), 2018
learning motivation is still low, so to overcome these problems required a learning model that is more focused on students to be more active in thinking on the ability to think creatively, one of which is assumed to improve the Creative Problem Solving ...
Yuda Purnama Putra
doaj  

CIIC Workshops for Creative Enterprises Potential Workshop Nominees.pdf

open access: yes, 2015
Business Model Workshops for Creative Enterprises - Information for Potential Workshop ...
Creative Industries Innovation Centre
core  

Metastasis on pause: How dormant tumor cells stay hidden within the tumor microenvironment and evade immune surveillance

open access: yesMolecular Oncology, EarlyView.
Dormant cancer cells can hide in distant organs for years, evading treatment and the immune system. This review highlights how signals from the surrounding tissue and immune environment keep these cells inactive or trigger their reawakening. Understanding these mechanisms may help develop therapies to eliminate or control dormant cells and prevent ...
Kanishka Tiwary   +1 more
wiley   +1 more source

Creative Commons

open access: yes, 2010
A link to the Creative Commons website, outlining all you need to know about attaching a licence to your open access materials.
Creative Commons
core  

Developmental programmes drive cellular plasticity, disease progression and therapy resistance in lung adenocarcinoma

open access: yesMolecular Oncology, EarlyView.
This study shows that lung adenocarcinomas exploit developmental branching morphogenesis to acquire a therapy resistant basal‐like tumour cell state. This process was found to be regulated by combined TP53 loss‐of‐function and type‐I interferon signalling, identifying a novel axis for biomarker and therapeutic target discovery.
Kamila J Bienkowska   +13 more
wiley   +1 more source

An exploration of the urban form preferences of Edmonton’s creative workers

open access: yes, 2017
Using data collected from the 2011 census and survey results, this study explores the urban form preferences of Edmonton’s creative workers. Edmonton continues to have a diverse economy, which includes a robust industrial sector.
Thomas, Craig Ronald
core  

Stimulator of interferon genes agonist augmented antitumor immunity of osimertinib in Egfr‐mutated lung cancer

open access: yesMolecular Oncology, EarlyView.
Combining osimertinib with the STING agonist ADU‐S100 activates innate and adaptive immunity to overcome the non‐inflamed microenvironment of Egfr‐mutant lung cancer. This combination increases NK and CD8+ T‐cell infiltration, associated with activation of the STING‐IRF3 pathway and local immunogenic cell death.
Jun Nishimura   +19 more
wiley   +1 more source

Loss of IGF‐1R impairs DNA‐PKcs recruitment to chromatin leading to defective end‐joining

open access: yesMolecular Oncology, EarlyView.
IGF‐1R promotes radioresistance by facilitating DNA‐PKcs recruitment to chromatin, enabling non‐homologous end‐joining (NHEJ) repair of double‐strand breaks. Inhibition or loss of IGF‐1R disrupts this recruitment to damage sites, driving compensatory reliance on microhomology‐mediated end‐joining (MMEJ) repair.
Matthew O. Ellis   +3 more
wiley   +1 more source

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