Results 141 to 150 of about 825,768 (324)

Quasi-Cyclic Low-Density Parity-Check Codes From Circulant Permutation Matrices [PDF]

, 2004
M.P.C. Fossorier
openalex   +1 more source

Extremal problems on permutations under cyclic equivalence

Discrete Mathematics, 1987
Let \(\sigma \in S_n\) be a permutation and let \([\sigma]\) be the class of all cyclic permutations of \(\sigma\). For \(\pi \in S_n\), \(\pi =(b_1,b_2,...,b_n)\), denote by \(\pi^R\) the permutation \((b_n,...,b_1)\in S_n\). Also denote by \(\) the set \([\sigma]\cup \{\tau^R;\quad \tau \in [\sigma]\}.\) In this paper the authors study the function \(
Nathan Linial, Paul Erdös, Shlomo Moran
openaire   +2 more sources

Unveiling unique protein and phosphorylation signatures in lung adenocarcinomas with and without ALK, EGFR, and KRAS genetic alterations

Molecular Oncology, EarlyView.
Proteomic and phosphoproteomic analyses were performed on lung adenocarcinoma (LUAD) tumors with EGFR, KRAS, or EML4–ALK alterations and wild‐type cases. Distinct protein expression and phosphorylation patterns were identified, especially in EGFR‐mutated tumors. Key altered pathways included vesicle transport and RNA splicing.
Fanni Bugyi, Mirjam Balbisi, Simon Sugár, Lóránd Váncza, Eszter Regős, Ilona Kovalszky, Ibolya Laczó, Tünde Harkó, Gábor Kecskeméti, Zoltán Szabó, Judit Moldvay, László Drahos, Lilla Turiák   +12 more
wiley   +1 more source

Investigating the cell of origin and novel molecular targets in Merkel cell carcinoma: a historic misnomer

Molecular Oncology, EarlyView.
This study indicates that Merkel cell carcinoma (MCC) does not originate from Merkel cells, and identifies gene, protein & cellular expression of immune‐linked and neuroendocrine markers in primary and metastatic Merkel cell carcinoma (MCC) tumor samples, linked to Merkel cell polyomavirus (MCPyV) status, with enrichment of B‐cell and other immune cell
Richie Jeremian, Sriraam Sivachandran, Melissa Galati, Brandon Ramchatesingh, Hibo Rijal, Johnny Hanna, Elena Netchiporouk, May Chergui, Margaret Redpath, Samy Abou Setah, Ivan V. Litvinov   +10 more
wiley   +1 more source

A synthetic benzoxazine dimer derivative targets c‐Myc to inhibit colorectal cancer progression

Molecular Oncology, EarlyView.
Benzoxazine dimer derivatives bind to the bHLH‐LZ region of c‐Myc, disrupting c‐Myc/MAX complexes, which are evaluated from SAR analysis. This increases ubiquitination and reduces cellular c‐Myc. Impairing DNA repair mechanisms is shown through proteomic analysis.
Nicharat Sriratanasak, Bodee Nutho, Worawat Wattanathana, Narumon Phaonakrop, Bunnatut Panasawatwong, Katharina Erlenbach‐Wuensch, Sittiruk Roytrakul, Regine Schneider‐Stock, Pithi Chanvorachote   +8 more
wiley   +1 more source

Quasi-cyclic extension based on PEG algorithm for construction of LDPC codes

Tongxin xuebao, 2008
Several important relations between the cycle structure on the Tanner graph and the parity-check matrix H of LDPC codes were proved.A new algorithm for constructing LDPC codes was proposed that introduced quasi-cyclic ex-tension method to PEG algorithm ...
LEI Jing1, WANG Jian-hui1, TANG Chao-jing1   +2 more
doaj   +2 more sources

A resilient image encryption scheme using Laplace transform

Egyptian Informatics Journal
The importance of image encryption in cryptography has surged due to advancement in image processing technology. Our research presents a novel image encryption technique using advanced image processing and permutation-based S-box generation.
Fariha Nawaz, Saba Inam, Shamsa Kanwal, Shaha Al-Otaibi, Fahima Hajjej   +4 more
doaj   +1 more source

Graphical representations of cyclic permutation groups

Discrete Applied Mathematics, 2020
Mariusz Grech, A. Kisielewicz
semanticscholar   +1 more source

Survivin and Aurora Kinase A control cell fate decisions during mitosis

Molecular Oncology, EarlyView.
Aurora A interacts with survivin during mitosis and regulates its centromeric role. Loss of Aurora A activity mislocalises survivin, the CPC and BubR1, leading to disruption of the spindle checkpoint and triggering premature mitotic exit, which we refer to as ‘mitotic slippage’.
Hana Abdelkabir, Shalitha Wickrama Arachchige, Sally P. Wheatley   +2 more
wiley   +1 more source

CDK11 inhibition induces cytoplasmic p21WAF1 splice variant by p53 stabilisation and SF3B1 inactivation

Molecular Oncology, EarlyView.
CDK11 inhibition stabilises the tumour suppressor p53 and triggers the production of an alternative p21WAF1 splice variant p21L, through the inactivation of the spliceosomal protein SF3B1. Unlike the canonical p21WAF1 protein, p21L is localised in the cytoplasm and has reduced cell cycle‐blocking activity.
Radovan Krejcir, Lukasz Arcimowicz, Lucia Martinkova, Vaclav Hrabal, Filip Zavadil Kokas, Tomas Henek, Martina Kucerikova, Ondrej Bonczek, Pavlina Zatloukalova, Lenka Hernychova, Philip J. Coates, Borivoj Vojtesek, David P. Lane   +12 more
wiley   +1 more source