Results 231 to 240 of about 206,171 (265)

Overcoming cytoplasmic incompatibility in Drosophila

Proceedings of the Royal Society of London Series B, Biological Sciences, 1998
The endocellular microbe Wolbachia pipientis infects a wide variety of invertebrate species, in which its presence is closely linked to a form of reproductive failure termed cytoplasmic incompatibility (CI). CI renders infected males unable to father offspring when mated to uninfected females.
Martin E Feder
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Wolbachia-Induced Cytoplasmic Incompatibility

2001
SYLVAIN CHARLAT, KOSTAS BOURTZIS 3 AND HERVE MERCOT 1 Institut Jacques Monod, Laboratoire Dynamique du Genome et Evolution, CNRS-Universites Paris 6, Paris 7, 2 place Jussieu, 75251 Paris Cedex 05, France, charlat@ijm.jussieu.fr 2 Department of Environmental and Natural Resources Management, University of Ioannina, Agrinio 30100, Greece 3 Insect ...
Charlat, S., Bourtzis, K., Merçot, H.
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Cytoplasmic incompatibility in insects

1997
Abstract Cytoplasmic incompatibility (CI) was first described in mosquitoes by Ghelelovitch (1952) and studied in detail by Laven in the 1950s (reviewed in Laven 1967b). Laven observed that crosses between different strains of mosquitoes sometimes failed to produce progeny, or produced progeny only when crossed in one direction (i.e ...
Ary A Hoffmann, Turelli Michael
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Cytoplasmic Incompatibility and Population Structure

Journal of Theoretical Biology, 1997
Wolbachiais a maternally inherited bacterial infection common in many insects. These bacteria cause cytoplasmic incompatibility, in which a cross between an infected male and an uninfected female is sterile. Infected females are always fertile, suggesting that an infected male produces a sterilizing product against which infected females are protected.
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On the dynamics of symbiote-dependent cytoplasmic incompatibility in culicine mosquitoes

Journal of Invertebrate Pathology, 1978
Abstract The population dynamics of cytoplasmic incompatibility in culicine mosquitoes is discussed in the context of recent discoveries of the biological basis of this phenomenon. A simple model is derived for predicting prevalence rates of infection with cytoplasmic incompatibility factors over successive generations of a host species.
Paul E M Fine
exaly   +3 more sources

Dynamics of Cytoplasmic Incompatability with MultipleWolbachiaInfections

Journal of Theoretical Biology, 1998
Wolbachia infections occur in many arthropods. These matrilineally inherited bacteria cause cytoplasmic incompatibility, in which a cross produces no offspring when between an infected male and an uninfected female. Some populations harbour multiple Wolbachia strains.
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The etiological agent of cytoplasmic incompatibility in Culex pipiens

Journal of Invertebrate Pathology, 1973
Abstract All individuals of Culex pipiens (wide sense) which have been examined were infected with Wolbachia pipientis . Larvae reared in tetracycline were freed of these symbiotes and remained free (aposymbiotic) in future generations. When males were freed of their symbiotes, they no longer displayed incompatibility.
J H, Yen, A R, Barr
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Cytoplasmic Incompatibility: Giant steps sideways

Current Biology, 1994
The horizontal transfer of a bacterial endosymbiont that is intimately associated with reproductive isolation in insects is now feasible and may, in principle, lead to new strategies for biological pest control.
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Cytoplasmic Incompatibility

2008
Beata Gabrys, John L. Capinera, Jesusa C. Legaspi, Benjamin C. Legaspi, Lewis S. Long, John L. Capinera, Jamie Ellis, Donald C. Weber, Pavel Saska, Caroline S. Chaboo, Linda wiener, James Cresswell, Donna Giberson, George M. Orphanides, John H. Klotz, Laurel D. Hansen, John L. Capinera, John B. Heppner, Derek S. Sikes, George Hangay, John B. Heppner, Peter Neuenschwander, Anthony C. Bellotti, John L. Capinera, Nancy C. Hinkle, Philip G. koehler, Steven J. Taylor, David Slaney, Philip Weinstein, Jun Mitsuhashi, John B. Heppner, Hilary Hurd, M. Patricia Juárez, John L. Capinera, Elizabeth A. Bernays, Heather J. McAuslane, Seiichi Moriya, Kelly Roe, Kenneth W. McCravy, Dakshina R. Seal, Waldemar Klassen, Efat Abou Fakhr Hammad, John L. Capinera, Meir Broza, James L. Nation, Maciej A. Pszczolkowski, Allen Sanborn, Robert Meagher, Marjorie A. Hoy, Daniel Potter, Allen Heath, John L. Capinera, Gary W. Bennett, Forrest W. Howard, Fernando E. Vega, Juan F. Barrera, David Rivers, Paul M. Choate, J. Howard Frank, Donald C. Weber, J. Howard Frank, John B. Heppner, Kenneth W. McCravy, Fabián D. Menalled, Douglas A. Landis, Andrei Sourakov, Thomas C. Emmel, Holly Downing, Lisa Neven, Elizabeth Mitcham, Seiji Tanaka, Ángeles Vázquez, James H. Tsai, John L. Capinera, John L. Capinera, James H. Tsai, John B. Heppner, John L. Capinera, John L. Capinera, O. E. Liburd, T. W. Nyoike, C. A. Scott, George W. Byers, John J. Herbert, Russell F. Mizell, Whitney Cranshaw, John B. Heppner, Hugh Smith, Loke T. Kok, Malcolm Edmunds, Takashi Okuda, George Hangay, Otto Merkl, Győző Szél, John All, Ron Cherry, Patrick De Clercq, Pierre Jolivet   +97 more
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Eradication of Culex pipiens fatigans through Cytoplasmic Incompatibility

Nature, 1967
Culex pipiens fatigans is the chief vector of filariasis in south-east Asia. Urbanization has often caused the numbers of this mosquito—and with it the danger of filariasis infection—to increase alarmingly1. The natural vigour, tolerance and fast development of resistance to insecticides of this mosquito necessitate the development of other control ...
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