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The association between deep vein thrombosis at admission and the time from injury to admission in hip fractures. [PDF]
Liu J, He M, Song R, Li J.
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Elevated lactate in diabetes activates the GPR132‐Src pathway in macrophages, inducing macrophage senescence and further enhancing foam cell formation. This mechanism exacerbates atherosclerotic progression. Abstract Diabetes is widely acknowledged as a significant risk factor for atherosclerosis, facilitating plaque formation through various ...
Xiaofeng Ge+10 more
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The effect of AngioJet thrombectomy combined with catheter-directed thrombolysis in acute deep vein thrombosis of the lower extremities. [PDF]
Xue J+5 more
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Risk factors for upper extremity deep vein thrombosis after esophagectomy for esophageal cancer in retrosternal reconstruction. [PDF]
Ogawa Y+6 more
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An artificial intelligence interpretable tool to predict risk of deep vein thrombosis after endovenous thermal ablation. [PDF]
Tabari A+6 more
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An unusual case of deep vein thrombosis of the upper extremity following breast reduction. [PDF]
Kazaz G, Yılmaz B, Saçak B.
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Action research on implementing the BOPPPS model in teaching mechanical prophylaxis techniques for deep vein thrombosis. [PDF]
Shen M, Wan P, Feng Z.
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The Lancet, 2005
Deep vein thrombosis and its sequelae pulmonary embolism and post-thrombotic syndrome are some of the most common disorders. A thrombus either arises spontaneously or is caused by clinical conditions including surgery, trauma, or prolonged bed rest. In these instances, prophylaxis with low-dose anticoagulation is effective.
S. Eichinger, Paul A. Kyrle
+9 more sources
Deep vein thrombosis and its sequelae pulmonary embolism and post-thrombotic syndrome are some of the most common disorders. A thrombus either arises spontaneously or is caused by clinical conditions including surgery, trauma, or prolonged bed rest. In these instances, prophylaxis with low-dose anticoagulation is effective.
S. Eichinger, Paul A. Kyrle
+9 more sources