Results 301 to 310 of about 1,179,077 (360)

Complement deficiency and disease

Immunology Today, 1991
Complement deficiency is associated with an increased prevalence of pyogenic infections and immune complex disease. The spectrum of disease in deficient individuals depends on the stage in the complement system at which the block in activation occurs. Here, Paul Morgan and Mark Walport review current knowledge of hereditary complement deficiencies in ...
B P, Morgan, M J, Walport
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A Deficiency Disease of Foxes

Science, 1940
A common and highly destructive dietary disease of silver foxes in captivity is pathologically the counterpart of Wernicke's hemorrhagic polioencephalitis of man. The disease in foxes is caused by feeding fish as 10 per cent., or more, of the diet.
R G, Green, C A, Evans
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Deficiency Diseases in the Caribbean

Journal of Interdisciplinary History, 1980
Deficiency Diseases in the Caribbean The historiography of slavery in the Americas has recently taken on a new biological dimension as historians have begun to appreciate the importance of pathogenic agents in any holistic understanding of their subject.
K F, Kiple, V H, Kiple
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Complement Deficiency and Disease

Australian and New Zealand Journal of Medicine, 1982
: The complement system consists of a series of serum proteins which interact to facilitate the action of certain classes of antibody and antigen/antibody complexes. Complement may be disturbed by pathological utilisation of one or both of its pathways or by a primary deficiency of an individual component.
J A, Charlesworth, B A, Pussell
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Complement deficiency and disease

Journal of Clinical Pathology, 2008
There are approximately 30 serum complement proteins (15% of the globulin fraction), excluding cell surface receptors, and regulatory proteins. Many are manufactured in the liver, and reduced complement is a feature of severe liver failure. Complement proteins contribute to the acute phase response, and high levels are seen in chronic untreated ...
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Trypsinogen deficiency disease

The Journal of Pediatrics, 1964
An infant with severe growth failure, hypoproteinemia, and edema is described. Clinical and laboratory studies indicate that this disorder results from an impaired capacity to hydrolyze ingested protein. By means of specific pancreatic proteolytic enzyme assays, it was established that the primary defect is a complete absence of trypsinogen. Absence of
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Complement deficiencies and human diseases

Annales de biologie clinique, 2014
The complement system is a complex system involving serum and membrane proteins interacting in a regulated manner. The complement system plays a major role in antibacterial immunity, in inflammation, and in immune complex processing. Therefore, deficiencies in complement proteins are associated with increased susceptibility to bacterial infections and ...
Jérémie, Rosain, Stéphanie, Ngo, Pauline, Bordereau, Nelly, Poulain, Stéphane, Roncelin, Paula, Vieira Martins, Marie-Agnès, Dragon-Durey, Véronique, Frémeaux-Bacchi   +7 more
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Complement deficiency and disease: An update

Molecular Immunology, 2006
Complement deficiencies are probably vastly under-diagnosed within clinical medicine. Judging from a Swedish study of C2 deficiency, a deficiency with an estimated prevalence of about 1/20,000 in Western countries, less than 10% of the deficiencies of the classical and alternative pathways and the late complement components are identified in Sweden. C1
A G, Sjöholm, G, Jönsson, J H, Braconier, G, Sturfelt, L, Truedsson   +4 more
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Zinc Deficiency in Crohn’s Disease

Digestion, 1977
Zinc nutriture has been studies in 30 patients with Crohn's disease using plasma zinc, hair zinc, and taste acuity as indices. Significant reductions in plasma zinc concentrations and taste acuity were seen in the patient groups as compared with normal controls.
N W, Solomons, I H, Rosenberg, H H, Sandstead, K P, Vo-Khactu   +3 more
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Zinc deficiency in Hodgkin's disease

European Journal of Cancer (1965), 1980
Abstract Serum, plasma, RBC and hair zinc concentrations were measured in children with Hodgkin's disease at diagnosis and during remission. Zinc values in all the measured compartments were found to be significantly low before treatment while serum, and plasma zinc levels returned to normal or above normal values during remission.
A O, Cavdar, E, Babacan, A, Arcasoy, J, Erten, U, Ertem   +4 more
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