Results 301 to 310 of about 1,030,121 (356)
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Postgraduate Medicine, 1973
In the past few decades, bedside observation and laboratory investigation have yielded important information concerning the modus operandi of the immune system. This article examines newer basic immunologic information, clinical features of the main immune deficiencies, and tests for investigation of these defects.
Gerald J. Gleich +2 more
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In the past few decades, bedside observation and laboratory investigation have yielded important information concerning the modus operandi of the immune system. This article examines newer basic immunologic information, clinical features of the main immune deficiencies, and tests for investigation of these defects.
Gerald J. Gleich +2 more
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Trypsinogen Deficiency Disease
Archives of Pediatrics & Adolescent Medicine, 1967TOWNES,1in 1965, reported a 6-week-old white male infant with chronic diarrhea, failure to gain weight, hypoproteinemia, and edema. The infant was unable to hydrolyze dietary protein due to a singular deficiency of pancreatic trypsinogen. The present report describes a second instance of this interesting defect.
D. A. Fisher, M. D. Morris
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Complement deficiency and disease
Immunology Today, 1991Complement deficiency is associated with an increased prevalence of pyogenic infections and immune complex disease. The spectrum of disease in deficient individuals depends on the stage in the complement system at which the block in activation occurs. Here, Paul Morgan and Mark Walport review current knowledge of hereditary complement deficiencies in ...
Mark Walport, B. Paul Morgan
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Science, 1940
A common and highly destructive dietary disease of silver foxes in captivity is pathologically the counterpart of Wernicke's hemorrhagic polioencephalitis of man. The disease in foxes is caused by feeding fish as 10 per cent., or more, of the diet.
Robert G. Green, C. A. Evans
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A common and highly destructive dietary disease of silver foxes in captivity is pathologically the counterpart of Wernicke's hemorrhagic polioencephalitis of man. The disease in foxes is caused by feeding fish as 10 per cent., or more, of the diet.
Robert G. Green, C. A. Evans
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Deficiency Diseases in the Caribbean
Journal of Interdisciplinary History, 1980Deficiency Diseases in the Caribbean The historiography of slavery in the Americas has recently taken on a new biological dimension as historians have begun to appreciate the importance of pathogenic agents in any holistic understanding of their subject.
Virginia H. Kiple, Kenneth F. Kiple
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Complement Deficiency and Disease
Australian and New Zealand Journal of Medicine, 1982: The complement system consists of a series of serum proteins which interact to facilitate the action of certain classes of antibody and antigen/antibody complexes. Complement may be disturbed by pathological utilisation of one or both of its pathways or by a primary deficiency of an individual component.
B. A. Pussell, J. A. Charlesworth
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Complement deficiency and disease
Journal of Clinical Pathology, 2008There are approximately 30 serum complement proteins (15% of the globulin fraction), excluding cell surface receptors, and regulatory proteins. Many are manufactured in the liver, and reduced complement is a feature of severe liver failure. Complement proteins contribute to the acute phase response, and high levels are seen in chronic untreated ...
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Iron deficiency and cardiovascular disease
Nature Reviews Cardiology, 2015Iron deficiency affects up to one-third of the world's population, and is particularly common in elderly individuals and those with certain chronic diseases. Iron excess can be detrimental in cardiovascular illness, and research has now also brought anaemia and iron deficiency into the focus of cardiovascular medicine.
Stefan D. Anker +4 more
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Trypsinogen deficiency disease
The Journal of Pediatrics, 1964An infant with severe growth failure, hypoproteinemia, and edema is described. Clinical and laboratory studies indicate that this disorder results from an impaired capacity to hydrolyze ingested protein. By means of specific pancreatic proteolytic enzyme assays, it was established that the primary defect is a complete absence of trypsinogen. Absence of
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COMPLEMENT DEFICIENCY AND DISEASE [PDF]
Rheumatology, 1993 Summary of complement deficiencies Component Number of cases Chromosome location of gene Disease associations Classical pathway proteins Clq Clr/Cls C4 C2 I>40 10 17 t>100 A, B chains 1,p; C chain ? Closely linked on 12,p13 Two genes C4A and C4B in MHC on chromosome 6 MHC locus on chromosome 6, adjacent to factor B Alternative pathway components Factor
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