Results 51 to 60 of about 23,869 (285)

Icariin Prevents Diabetes-Induced Bone Loss in Rats by Reducing Blood Glucose and Suppressing Bone Turnover

open access: yesMolecules, 2019
Diabetic Osteoporosis (DOP) is a common metabolic bone disease, characterized by decreased bone mineral density (BMD) and destruction of bone microstructure.
Shanshan Qi   +4 more
doaj   +1 more source

Table_1_Identification of ferroptosis-related molecular clusters and genes for diabetic osteoporosis based on the machine learning.xlsx

open access: yes, 2023
BackgroundDiabetic osteoporosis exhibits heterogeneity at the molecular level. Ferroptosis, a controlled form of cell death brought on by a buildup of lipid peroxidation, contributes to the onset and development of several illnesses.
Lei Meng (1394350)   +12 more
core   +1 more source

The role of wnt signaling in diabetes-induced osteoporosis

open access: yesDiabetology & Metabolic Syndrome, 2023
Osteoporosis, a chronic complication of diabetes mellitus, is characterized by a reduction in bone mass, destruction of bone microarchitecture, decreased bone strength, and increased bone fragility.
Kairan Bao   +4 more
doaj   +1 more source

Table_5_Identification of ferroptosis-related molecular clusters and genes for diabetic osteoporosis based on the machine learning.xlsx

open access: yes, 2023
BackgroundDiabetic osteoporosis exhibits heterogeneity at the molecular level. Ferroptosis, a controlled form of cell death brought on by a buildup of lipid peroxidation, contributes to the onset and development of several illnesses.
Lei Meng (1394350)   +12 more
core   +1 more source

Catalpol Protects Against High Glucose-Induced Bone Loss by Regulating Osteoblast Function

open access: yesFrontiers in Pharmacology, 2021
Objective: The overall objective of this study was to investigate the effects of catalpol on bone remodeling of diabetic osteoporosis by regulating osteoblast differentiation and migration.Method: Using a murine model of diabetic osteoporosis, to detect ...
Lu Zhao   +7 more
doaj   +1 more source

Type I diabetic osteoporosis and osteoblast apoptosis

open access: yes, 2012
TYPE I DIABETIC OSTEOPOROSIS AND OSTEOBLAST APOPTOSIS ByLindsay Martin CoeType I diabetes is a metabolic disorder that affects roughly 1 million people in the United States.
Coe, Lindsay Martin
core   +1 more source

Image3_PPARβ/δ Agonist Alleviates Diabetic Osteoporosis via Regulating M1/M2 Macrophage Polarization.pdf

open access: yes, 2021
Diabetic osteoporosis is a common complication in diabetic patients, leading to increased fracture risk and impaired bone healing. As a member of the peroxisome proliferator-activated receptor (PPAR) family, PPARβ/δ agonist is suggested as a therapeutic ...
Weimin Lin (2117626)   +4 more
core   +1 more source

Type 2 diabetes is associated with higher trabecular bone density but lower cortical bone density: the Vietnam Osteoporosis Study

open access: yes, 2018
© 2018, International Osteoporosis Foundation and National Osteoporosis Foundation. Summary: It is not clear why type 2 diabetes (T2D) has an increased risk of fracture despite higher areal bone mineral density.
Ho-Pham, LT   +4 more
core   +1 more source

Image2_PPARβ/δ Agonist Alleviates Diabetic Osteoporosis via Regulating M1/M2 Macrophage Polarization.pdf

open access: yes, 2021
Diabetic osteoporosis is a common complication in diabetic patients, leading to increased fracture risk and impaired bone healing. As a member of the peroxisome proliferator-activated receptor (PPAR) family, PPARβ/δ agonist is suggested as a therapeutic ...
Weimin Lin (2117626)   +4 more
core   +1 more source

Image1_PPARβ/δ Agonist Alleviates Diabetic Osteoporosis via Regulating M1/M2 Macrophage Polarization.pdf

open access: yes, 2021
Diabetic osteoporosis is a common complication in diabetic patients, leading to increased fracture risk and impaired bone healing. As a member of the peroxisome proliferator-activated receptor (PPAR) family, PPARβ/δ agonist is suggested as a therapeutic ...
Weimin Lin (2117626)   +4 more
core   +1 more source

Home - About - Disclaimer - Privacy