Results 1 to 10 of about 323 (80)
Some of the next articles are maybe not open access.
Disease-specific oligodendrocyte lineage cells arise in multiple sclerosis
Nature Medicine, 2018David van Bruggen +2 more
exaly
Interactions between genetic, lifestyle and environmental risk factors for multiple sclerosis
Nature Reviews Neurology, 2016Tomas Olsson +2 more
exaly
Remyelination therapies: a new direction and challenge in multiple sclerosis
Nature Reviews Drug Discovery, 2017Jason R Plemel, Voon Wee Yong
exaly
Fingolimod (FTY720): discovery and development of an oral drug to treat multiple sclerosis
Nature Reviews Drug Discovery, 2010exaly
Diffuse Cerebral Sclerosis of Schilder [PDF]
National Cancer Institute
openalex +2 more sources
Defining the natural history of tumefactive demyelination: A retrospective cohort of 257 patients
Abstract Objective To describe demographic, clinical, and radiographic features of tumefactive demyelination (TD) and identify factors associated with severe attacks and poor outcomes. Methods Retrospective review of TD cases seen at Mayo Clinic, 1990–2021.
Mahboubeh Fereidan‐Esfahani +8 more
wiley +1 more source
In this lecture, multiple sclerosis variants will be discussed, which include Solitary Sclerosis, Schilder's disease, Baló's concentric sclerosis, Marburg variant of multiple sclerosis.
Ebrahim Kouchaki
doaj
Silent Tumefactive Demyelinating Lesions and Radiologically Isolated Syndrome
Demyelinating lesions larger than 2 cm in diameter, with or without edema, are known as tumefactive demyelinating lesions (TDLs). They constitute a rare inflammatory demyelinating disorder of the central nervous system. TDLs are typically characterized by headaches, cortical symptoms such as aphasia, hemiparesis, hemisensory deficits, seizures, and ...
Ozgul Ekmekci +2 more
wiley +1 more source
A Case of Acute Disseminated Encephalomyelitis in a Middle‐Aged Adult
Objectives. Acute disseminated encephalomyelitis (ADEM) is an inflammatory demyelinating disorder that is often preceded by infection or recent vaccination. Encephalopathy and focal neurological deficits are usually manifest several weeks after a prodromal illness with rapidly progressive neurologic decline.
Nicole Mahdi +4 more
wiley +1 more source

