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Current Opinion in Hematology, 1997
Fibrinogen abnormalities can be classified as congenital or acquired. Each class manifests quantitative or qualitative alterations; the latter are known as dysfibrinogenemias. In dysfibrinogenemias, structural defects cause alterations in the conversion of fibrinogen to fibrin.
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Fibrinogen abnormalities can be classified as congenital or acquired. Each class manifests quantitative or qualitative alterations; the latter are known as dysfibrinogenemias. In dysfibrinogenemias, structural defects cause alterations in the conversion of fibrinogen to fibrin.
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Sneddon Syndrome revealing dysfibrinogenemia
International Journal of Dermatology, 2003Sneddon syndrome (SS) is the association of cerebral ischemic events and livedo and is caused by vascular thrombosis. It is often associated with a primitive antiphospholipid syndrome (PAPS). In other cases, several thrombophilic conditions have been implicated. Dysfibrinogenemia is a qualitative defect of fibrinogen.
Khosrotehrani, Kiarash +4 more
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A Hereditary Dysfibrinogenemia: Fibrinogen Awaji
Haemostasis, 2009Abnormal function of fibrinogen was observed in a 2 5-year-old woman with no symptoms attributable to dysfibrinogenemia. Disturbed polymerization of fibrin monomer was identified, but the release of fibrinopeptide from the purified fibrinogen and the cross-linking by factor XIII were normal.
T, Matsuo +5 more
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Dysfibrinogenemia in Obstructive Liver Disease
Journal of Pediatric Gastroenterology and Nutrition, 1987SummaryAcquired dysfibrinogenemia was documented in a 4‐year‐old child with obstructive jaundice of 1‐month duration, secondary to a choledochal cyst involving the distal common bile duct. It was characterized by decreased thrombin coagulable protein with elevated immunoassayable fibrinogen resulting in abnormal thrombin and reptilase times.
J, Levy, M J, Pettei, J I, Weitz
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Congenital dysfibrinogenemia : Fibrinogen lille
Thrombosis Research, 1978A dysfibrinogenemia (fibrinogen Lille) was detected in a 6 year old girl with no history of unusual bleeding. Thrombin and batroxobin (“reptilase”) times were markedly prolonged. The plasma fibrinogen level was unmeasurable by a kinetic method (Clauss), whereas normal values were obtained by immunochemical (Laurell) and gravimetric (Ingram) assays ...
M H, Denninger +5 more
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Management of pregnancy in dysfibrinogenemia cases
Blood Coagulation & Fibrinolysis, 2017Dysfibrinogenemia is a very challenging disorder, and there are no firm guidelines on treatment for pregnant patients with dysfibrinogenemia. A 37-year-old patient with a history of six unexplained recurrent miscarriages was referred for thrombophilia testing.
Rucha, Patil +3 more
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Dysfibrinogenemia and thrombosis.
Nouvelle revue francaise d'hematologie, 1992A thrombotic tendency (venous or arterial) has been reported in some cases of dysfibrinogenemia. We report here the mechanism by which these thrombosis may occur. It may be related either to a defective clot lysis due to a poor reactivity toward fibrinolytic enzymes or to a defective thrombin binding capacity of the abnormal clot.
J, Soria +5 more
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[Dysfibrinogenemia. A new case: dysfibrinogenemia Giessen III (author's transl)].
Klinische Wochenschrift, 1977A new case of dysfibrinogenemia is reported which shows no signs of a haermorrhagic diathesis (dysfibrinogenemia Giessen III). The abnormal fibrinogen was detected by only slight but characteristic alterations of some parameter of the coagulation analysis (prolonged clotting times after addition of thrombin, Reptilase and thrombin coagulase; low ...
F R, Matthias +4 more
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Dysfibrinogenemia and placental abruption
Obstetrics & Gynecology, 2000R Z, Edwards, A, Rijhsinghani
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