Results 131 to 140 of about 1,246 (182)
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Dysfibrinogenemia and Thrombosis
Seminars in Thrombosis and Hemostasis, 1999Congenital abnormal fibrinogen molecules (dysfibrinogenemias) are due to structural defects in the molecule. The molecular structure of the fibrinogen molecule is to a great extent known and this has allowed identification of the abnormalities at a molecular level.
Michael W Mosesson, M W Mosesson
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Congenital Dysfibrinogenemia Presented with Massive Hematomas Formed after Hysterectomy [PDF]
Case Reports in Clinical Medicine, 2021 ArticleCase Reports in Clinical Medicine.
Takuji Tanaka, Nobuo Okumura
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Acute obstetric coagulopathy during postpartum hemorrhage is caused by hyperfibrinolysis and dysfibrinogenemia: an observational cohort study [PDF]
Journal of Thrombosis and Haemostasis, 2023 Background Postpartum hemorrhage (PPH) may be exacerbated by hemostatic impairment. Information about PPH-associated coagulopathy is limited, often resulting in treatment strategies based on data derived from trauma studies.
Rachel E Collis
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Dysfibrinogenemia: from molecular anomalies to clinical manifestations and management
Journal of Thrombosis and Haemostasis, 2015 Congenital dysfibrinogenemia is a qualitative congenital fibrinogen disorder characterized by normal antigen levels of a dysfunctional fibrinogen. The diagnosis is usually based on discrepancies between fibrinogen activity and antigen levels, but could ...
Alessandro Casini +1 more
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Congenital dysfibrinogenemias. A review
La Ricerca in Clinica e in Laboratorio, 1985Inherited qualitative abnormalities of fibrinogen have been documented in 144 families. These dysfibrinogenemias have been inherited as autosomal dominant traits and usually are clinically silent, but in some cases are associated with bleeding, thrombosis, or defective wound healing. Dysfibrinogenemias may be associated with defects in any of the three
E, Rocha +4 more
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Current Opinion in Hematology, 1997
Fibrinogen abnormalities can be classified as congenital or acquired. Each class manifests quantitative or qualitative alterations; the latter are known as dysfibrinogenemias. In dysfibrinogenemias, structural defects cause alterations in the conversion of fibrinogen to fibrin.
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Fibrinogen abnormalities can be classified as congenital or acquired. Each class manifests quantitative or qualitative alterations; the latter are known as dysfibrinogenemias. In dysfibrinogenemias, structural defects cause alterations in the conversion of fibrinogen to fibrin.
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A dysfibrinogenemia leading to resistance to bovine thrombin
Acta Clinica Belgica, 2014 Introduction: A 26-year-old woman presented to our institute for a routine check-up. Nothing was abnormal excepted a prolonged Thrombin Time and a low fibrinogen concentration determined by the Clauss method. Fibrinogen concentration was then measured by
Depoorter, Maxime, Eeckhoudt, Stéphane
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A Hereditary Dysfibrinogenemia: Fibrinogen Awaji
Haemostasis, 2009Abnormal function of fibrinogen was observed in a 2 5-year-old woman with no symptoms attributable to dysfibrinogenemia. Disturbed polymerization of fibrin monomer was identified, but the release of fibrinopeptide from the purified fibrinogen and the cross-linking by factor XIII were normal.
T, Matsuo +5 more
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Dysfibrinogenemia in Obstructive Liver Disease
Journal of Pediatric Gastroenterology and Nutrition, 1987SummaryAcquired dysfibrinogenemia was documented in a 4‐year‐old child with obstructive jaundice of 1‐month duration, secondary to a choledochal cyst involving the distal common bile duct. It was characterized by decreased thrombin coagulable protein with elevated immunoassayable fibrinogen resulting in abnormal thrombin and reptilase times.
J, Levy, M J, Pettei, J I, Weitz
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