Results 351 to 360 of about 414,897 (392)
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Chronic Traumatic Encephalopathy

Seminars in neurology, 2020
mental risk factors has often been a suspected trigger for the development of neurodegenerative disease. Yet of all the possible environmental risk factors put forth, trauma to the central nervous system is one of the most consistent candi dates for ...
Jesse B. Mez, R. Stern, A. Mckee
semanticscholar   +1 more source

Neonatal Encephalopathy: Beyond Hypoxic-Ischemic Encephalopathy.

NeoReviews, 2021
Neonatal encephalopathy is a clinical syndrome of neurologic dysfunction that encompasses a broad spectrum of symptoms and severity, from mild irritability and feeding difficulties to coma and seizures.
Jeffrey B. Russ   +2 more
semanticscholar   +1 more source

Neonatal encephalopathy following fetal distress. A clinical and electroencephalographic study.

Archives of Neurology, 1976
Twenty-one neonates of over 36 weeks' gestation suffered perinatal asphyxia but not chronic hypoxia. Three clinical stages of postanoxic encephalopathy were distinguished.
H. Sarnat, M. S. Sarnat
semanticscholar   +1 more source

Glycine Encephalopathy

Neuropediatrics, 1979
4 cases of nonketotic hyperglycinemia (glycine encephalopathy), one with autopsy, are presented and the literature on 61 cases is reviewed. Major clinical signs include early hypotonia, lethargy and erratic and massive myoclonias with respiratory distrubances, starting during the first days of life after a symptom-free interval.
DALLA BERNARDINA, Bernardo   +3 more
openaire   +3 more sources

CYCLOLEUCINE ENCEPHALOPATHY

Journal of Neuropathology and Experimental Neurology, 1978
Greco C. M., Powell H. C, Garrett R. S. & Lampert P. W. (1980) Neuropathology and Applied Neurobiology 6,349–360Cycloleucine encephalopathyCycloleucine, a non‐metabolizable amino acid analogue produces status spongiosus in cerebral white matter of rats and mice as well as a distinctive lesion of astrocytes.
C M, Greco   +3 more
openaire   +2 more sources

Neonatal encephalopathy and hypoxic–ischemic encephalopathy

2019
Acute hypoxic-ischemic encephalopathy around the time of birth remains a major cause of death and life-long disability. The key insight that led to the modern revival of studies of neuroprotection was that, after profound asphyxia, many brain cells show initial recovery from the insult during a short "latent" phase, typically lasting approximately 6h ...
Gunn, A, Thoresen, Marianne
openaire   +4 more sources

Important Unresolved Questions in the Management of Hepatic Encephalopathy: An ISHEN Consensus.

American Journal of Gastroenterology, 2020
Management of hepatic encephalopathy (HE) remains challenging from a medical and psychosocial perspective. Members of the International Society for Hepatic Encephalopathy and Nitrogen Metabolism recognized 5 key unresolved questions in HE management ...
J. Bajaj   +11 more
semanticscholar   +1 more source

Hepatic Encephalopathy

2011
Hepatic encephalopathy (HE) is a neuropsychiatric syndrome which can develop in the course of chronic and acute liver disease. It is characterized by cognitive and motoric deficits of varying severity. HE is functional in nature, potentially reversible and is thought to reflect the clinical manifestation of a low-grade cerebral edema, which exacerbates
openaire   +4 more sources

Chronic Traumatic Encephalopathy

Traumatic Brain Injury, 2019
Chronic traumatic encephalopathy (CTE) is a progressive, neurodegenerative tauopathy associated with exposure to repetitive head injury, including concussions and subconcussions.
Charles J. Golden, M. Zusman
semanticscholar   +1 more source

Hypoxic-ischemic encephalopathy: a review for the clinician.

JAMA pediatrics, 2015
IMPORTANCE Hypoxic-ischemic encephalopathy (HIE) occurs in 1 to 8 per 1000 live births in developed countries. Historically, the clinician has had little to offer neonates with HIE other than systemic supportive care.
M. Douglas-Escobar, M. Weiss
semanticscholar   +1 more source

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