Results 351 to 360 of about 381,364 (391)
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Neuropediatrics, 1979
4 cases of nonketotic hyperglycinemia (glycine encephalopathy), one with autopsy, are presented and the literature on 61 cases is reviewed. Major clinical signs include early hypotonia, lethargy and erratic and massive myoclonias with respiratory distrubances, starting during the first days of life after a symptom-free interval.
DALLA BERNARDINA, Bernardo +3 more
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4 cases of nonketotic hyperglycinemia (glycine encephalopathy), one with autopsy, are presented and the literature on 61 cases is reviewed. Major clinical signs include early hypotonia, lethargy and erratic and massive myoclonias with respiratory distrubances, starting during the first days of life after a symptom-free interval.
DALLA BERNARDINA, Bernardo +3 more
openaire +3 more sources
Journal of Neuropathology and Experimental Neurology, 1978
Greco C. M., Powell H. C, Garrett R. S. & Lampert P. W. (1980) Neuropathology and Applied Neurobiology 6,349–360Cycloleucine encephalopathyCycloleucine, a non‐metabolizable amino acid analogue produces status spongiosus in cerebral white matter of rats and mice as well as a distinctive lesion of astrocytes.
C M, Greco +3 more
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Greco C. M., Powell H. C, Garrett R. S. & Lampert P. W. (1980) Neuropathology and Applied Neurobiology 6,349–360Cycloleucine encephalopathyCycloleucine, a non‐metabolizable amino acid analogue produces status spongiosus in cerebral white matter of rats and mice as well as a distinctive lesion of astrocytes.
C M, Greco +3 more
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Important Unresolved Questions in the Management of Hepatic Encephalopathy: An ISHEN Consensus.
American Journal of Gastroenterology, 2020Management of hepatic encephalopathy (HE) remains challenging from a medical and psychosocial perspective. Members of the International Society for Hepatic Encephalopathy and Nitrogen Metabolism recognized 5 key unresolved questions in HE management ...
J. Bajaj +11 more
semanticscholar +1 more source
Neonatal encephalopathy and hypoxic–ischemic encephalopathy
2019Acute hypoxic-ischemic encephalopathy around the time of birth remains a major cause of death and life-long disability. The key insight that led to the modern revival of studies of neuroprotection was that, after profound asphyxia, many brain cells show initial recovery from the insult during a short "latent" phase, typically lasting approximately 6h ...
Gunn, A, Thoresen, Marianne
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Neonatal encephalopathy following fetal distress. A clinical and electroencephalographic study.
Archives of Neurology, 1976Twenty-one neonates of over 36 weeks' gestation suffered perinatal asphyxia but not chronic hypoxia. Three clinical stages of postanoxic encephalopathy were distinguished.
H. Sarnat, M. S. Sarnat
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Chronic Traumatic Encephalopathy
Traumatic Brain Injury, 2019Chronic traumatic encephalopathy (CTE) is a progressive, neurodegenerative tauopathy associated with exposure to repetitive head injury, including concussions and subconcussions.
Charles J. Golden, M. Zusman
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2011
Hepatic encephalopathy (HE) is a neuropsychiatric syndrome which can develop in the course of chronic and acute liver disease. It is characterized by cognitive and motoric deficits of varying severity. HE is functional in nature, potentially reversible and is thought to reflect the clinical manifestation of a low-grade cerebral edema, which exacerbates
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Hepatic encephalopathy (HE) is a neuropsychiatric syndrome which can develop in the course of chronic and acute liver disease. It is characterized by cognitive and motoric deficits of varying severity. HE is functional in nature, potentially reversible and is thought to reflect the clinical manifestation of a low-grade cerebral edema, which exacerbates
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Pathophysiology of Encephalopathy
2000The metabolic complexity of the central nervous system (CNS) makes it dependent upon the functional integrity of other body systems for the adequate provision of essential nutrients and elimination of toxins. It is therefore not surprising that various metabolic effects on the CNS are secondary to systemic diseases.
LATRONICO, Nicola +2 more
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Annals of the New York Academy of Sciences, 2008
Hashimoto's encephalopathy (HE) is a controversial neurological disorder that comprises a heterogenous group of neurological symptoms that manifest in patients with high titers of antithyroid antibodies. Clinical manifestations of HE may include encephalopathic features such as seizures, behavioral and psychiatric manifestations, movement disorders ...
Carlos A. Pardo, Nicoline Schiess
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Hashimoto's encephalopathy (HE) is a controversial neurological disorder that comprises a heterogenous group of neurological symptoms that manifest in patients with high titers of antithyroid antibodies. Clinical manifestations of HE may include encephalopathic features such as seizures, behavioral and psychiatric manifestations, movement disorders ...
Carlos A. Pardo, Nicoline Schiess
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Hypoxic-ischemic encephalopathy: a review for the clinician.
JAMA pediatrics, 2015IMPORTANCE Hypoxic-ischemic encephalopathy (HIE) occurs in 1 to 8 per 1000 live births in developed countries. Historically, the clinician has had little to offer neonates with HIE other than systemic supportive care.
M. Douglas-Escobar, M. Weiss
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