Results 331 to 340 of about 24,309,158 (381)
YAP signaling orchestrates the endothelin-1-guided invadopodia formation in high-grade serous ovarian cancer. [PDF]
Tocci P +5 more
europepmc +1 more source
Prediction of Japan Score and Society of Thoracic Surgeons Risk Scores for Patients Undergoing Cardiovascular Surgery by Serum Growth Differentiation Factor-15 and Endothelin-1 Levels. [PDF]
Fukuda T +9 more
europepmc +1 more source
The dual endothelin-1 antagonist aprocitentan alleviates mitochondrial oxidative stress in human cardiac fibroblasts. [PDF]
Varzideh F +6 more
europepmc +1 more source
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Journal of Cardiovascular Pharmacology, 1993
Sepsis leads to changes in blood volume and distribution. In the experiments reported here we monitored circulatory changes during endotoxemia and their relation to portal and systemic levels of endothelin-1 (ET-1). Piglets were monitored cardiovascularly under ketamine anesthesia for 6 h after an endotoxin infusion (saline controls).
U, Myhre +3 more
openaire +2 more sources
Sepsis leads to changes in blood volume and distribution. In the experiments reported here we monitored circulatory changes during endotoxemia and their relation to portal and systemic levels of endothelin-1 (ET-1). Piglets were monitored cardiovascularly under ketamine anesthesia for 6 h after an endotoxin infusion (saline controls).
U, Myhre +3 more
openaire +2 more sources
Endothelin-1-induced nociception
Life Sciences, 1991Intracerebroventricular (i.c.v.) or intrathecal (i.t.) administration of morphine to mice antagonized the abdominal constriction induced by an i.p. injection of endothelin-1 (ET-1; 0.1 mg/kg). The ED50 values (95% confidence intervals) were 39.3 (16.5-80.2) ng and 1.5 (0.8-4.9) ng, respectively.
R B, Raffa +3 more
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Endothelin-1 and tumour development
European Journal of Surgical Oncology (EJSO), 1998There has been a growing conviction amongst oncologists that cancer is a disease characterized by changes in specific molecules. These changes include alteration in the structure, regulation or quantity of growth factors and their receptors, signal transducers and the proteins encoded by dominant or suppressor/recessive oncogenes.
E H, Asham, M, Loizidou, I, Taylor
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