Results 91 to 100 of about 100,569 (330)
Placental protein 13 (PP13) induces hypotension in rats. This study aims to evaluate PP13 effects on isolated uterine arteries from nonpregnant and mid-pregnant rats.
Tijana Drobnjak+6 more
doaj +1 more source
Coronary heart disease (CHD) is a major cause of mortality. Morphological substrate of CHD in most cases is atherosclerosis, which is based on structural genes polymorphism eNOS and AGTR2.
A. L. Khokhlov+4 more
doaj +1 more source
The Characteristics and Regulatory Mechanisms of Superoxide Generation from eNOS Reductase Domain. [PDF]
In addition to superoxide (O2.-) generation from nitric oxide synthase (NOS) oxygenase domain, a new O2.- generation site has been identified in the reductase domain of inducible NOS (iNOS) and neuronal NOS (nNOS).
Hu Peng+4 more
doaj +1 more source
Bahaman patch reefs: Numerous and neglected
Abstract Phanerozoic patch reefs are extensively studied because they are abundant and are potential reservoirs for valuable fluids. In contrast, geologic studies of modern reefs have largely focused on platform margins, leaving patch reefs comparatively neglected. What conditions favour patch‐reef development?
Paul Enos, Clay Robertson
wiley +1 more source
Ang II-induced contraction is impaired in the aortas of renovascular hypertensive animal model
Renin–angiotensin system plays a critical role in blood pressure control, and the abnormal activation of the AT1 receptor contributes to the development of renovascular hypertension.
Bernah M Fahning+4 more
doaj +1 more source
Activation of thromboxane A2 receptors mediates endothelial dysfunction in diabetic mice
Background: Diabetes is one of high-risk factors for cardiovascular disease. Improvement of endothelial dysfunction in diabetes reduces vascular complications. However, the underlying mechanism needs to be uncovered. This study was conducted to elucidate
Xiaona Xie+5 more
doaj +1 more source
Serine 1179 Phosphorylation of Endothelial Nitric Oxide Synthase Increases Superoxide Generation and Alters Cofactor Regulation. [PDF]
Endothelial nitric oxide synthase (eNOS) is responsible for maintaining systemic blood pressure, vascular remodeling and angiogenesis. In addition to producing NO, eNOS can also generate superoxide (O2-.) in the absence of the cofactor ...
Hu Peng+5 more
doaj +1 more source
Norfuraneol dephosphorylates eNOS at threonine 495 and enhances eNOS activity in human endothelial cells [PDF]
Pentoses are widely abundant in organic food. Thermal treatment of pentoses leads to the formation of norfuraneol (NF). The aim of this study was to show whether NF, which is taken up regularly, for example with cooked food, affects the human endothelial nitric oxide synthase (eNOS) system.The study was performed using cultured human umbilical vein ...
Schmitt, Christoph A.+4 more
openaire +4 more sources
Regarding cardiovascular effects, DMH decreased vascular reactivity to phenylephrine and acetylcholine. We observed that the combination of DPH and 8‐Cl‐T was identified as the responsible for increasing blood pressure and heart rate in the biological model used.
Jazmín Flores‐Monroy+3 more
wiley +1 more source
AIMS The role of endothelial nitric oxide synthase (eNOS)/NO signalling is well documented in late ischaemic preconditioning (IPC); however, the role of eNOS and its activation in early IPC remains controversial.
Changjun Yang+4 more
semanticscholar +1 more source