Results 261 to 270 of about 100,569 (330)

eNOS T-786C Genotype, Physical Activity, and Peak Forearm Blood Flow in Females

open access: bronze, 2003
SHERI A. DATA   +5 more
openalex   +1 more source

Oxidative Stress: Signaling Pathways, Biological Functions, and Disease

open access: yesMedComm, Volume 6, Issue 7, July 2025.
Oxidative stress causes cellular damage across multiple systems, contributing to neurodegeneration (Alzheimer's, Parkinson's, Huntington's), cancer progression and resistance, cardiovascular diseases (atherosclerosis, heart failure), liver and kidney injury, metabolic disorders (diabetes, obesity), autoimmune diseases, musculoskeletal decline, retinal ...
Sixuan Liu   +4 more
wiley   +1 more source

Investigation of PDE5 effect on NOS in nasal polyp pathophysiology. [PDF]

open access: yesEur Arch Otorhinolaryngol
Mutlu V   +4 more
europepmc   +1 more source

Lower Skeletal Muscle Nutritive Blood Flow in Older Women Is Related to eNOS Protein Content

open access: bronze, 2003
Robert C. Hickner   +4 more
openalex   +1 more source

Lactate Metabolism and Lactylation Modification: New Opportunities and Challenges in Cardiovascular Disease

open access: yesMedComm, Volume 6, Issue 7, July 2025.
Lactate, beyond a metabolic byproduct, regulates immunity and inflammation, linked to cardiovascular diseases (CVD) via metabolic reprogramming. Lactylation, a lactate‐driven epigenetic mark, modulates gene expression in fibrosis, lipid disorders, and CVD progression.
Mengyang Song   +3 more
wiley   +1 more source

Males and females respond differently to treatment during isolated kidney perfusion: combined effects of glucocorticoid and estradiol. [PDF]

open access: yesFront Transplant
Vidal-Dos-Santos M   +9 more
europepmc   +1 more source

S-glutathionylation uncouples eNOS and regulates its cellular and vascular function

open access: yesNature, 2010
Chun-an Chen   +10 more
semanticscholar   +1 more source

Specialized Pro‐Resolving Mediators as Emerging Players in Cardioprotection: From Inflammation Resolution to Therapeutic Potential

open access: yesActa Physiologica, Volume 241, Issue 7, July 2025.
ABSTRACT Aim Timely myocardial reperfusion is essential for restoring blood flow to post‐ischemic tissue, thereby reducing cardiac injury and limiting infarct size. However, this process can paradoxically result in additional, irreversible myocardial damage, known as myocardial ischemia–reperfusion injury (MIRI).
Anna De Bartolo   +3 more
wiley   +1 more source

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