Results 141 to 150 of about 582,301 (339)

Mitochondria‐associated membranes (MAMs): molecular organization, cellular functions, and their role in health and disease

FEBS Open Bio, EarlyView.
Mitochondria‐associated membranes (MAMs) are contact sites between the endoplasmic reticulum and mitochondria that regulate calcium signaling, lipid metabolism, autophagy, and stress responses. This review outlines their molecular organization, roles in cellular homeostasis, and how dysfunction drives neurodegeneration, metabolic disease, cancer, and ...
Viet Bui, Maryline Santerre, Natalia Shcherbik, Bassel E Sawaya   +3 more
wiley   +1 more source

Excretion of Antimony [PDF]

BMJ, 1948
openaire   +3 more sources

Association of urinary sodium and potassium excretion with blood pressure.

New England Journal of Medicine, 2014
A. Mente, M. O’Donnell, S. Rangarajan, M. McQueen, P. Poirier, A. Wielgosz, H. Morrison, Wei Li, Xingyu Wang, C. Di, P. Mony, A. Devanath, A. Rosengren, A. Oguz, K. Zatońska, A. Yusufali, P. López-Jaramillo, Á. Avezum, Noorhassim Ismail, F. Lanas, T. Puoane, R. Diaz, R. Kelishadi, R. Iqbal, R. Yusuf, J. Chifamba, R. Khatib, K. Teo, S. Yusuf   +28 more
semanticscholar   +1 more source

Tryptophan metabolite atlas uncovers organ, age, and sex‐specific variations

FEBS Open Bio, EarlyView.
Tryptophan metabolites were analyzed across twelve organs, the central nervous system, and serum in male and female mice at three life stages. We found tissue‐, sex‐, and age‐specific differences, including increased indole‐3‐pyruvate and kynurenine in aging males.
Lizbeth Perez‐Castro, Afshan F. Nawas, Jessica A. Kilgore, Pedro A. S. Nogueira, M.Carmen Lafita‐Navarro, Paul H. Acosta, Roy Garcia, Noelle S. Williams, Maralice Conacci‐Sorrell   +8 more
wiley   +1 more source

Porphyrin Excretion following Antipyretics [PDF]

, 1939
George G. Brownlee
openalex   +1 more source

Urinary Albumin Excretion Predicts Cardiovascular and Noncardiovascular Mortality in General Population

Circulation, 2002
H. Hillege, V. Fidler, G. Diercks, W. V. van Gilst, D. de Zeeuw, D. V. van Veldhuisen, R. Gans, W. Janssen, D. Grobbee, P. D. de Jong   +9 more
semanticscholar   +1 more source

Lactoferrin treatment activates acetylcholinesterase, decreasing acetylcholine levels in non‐small cell lung cancer (NSCLC) cell culture supernatants, inhibiting cell survival

FEBS Open Bio, EarlyView.
Representation of the suggested mode of action of lactoferrin (Lf) in nonsmall cell lung cancer (NSCLC) A549 cells. Lf induces activation of caspase‐3 by activating p53 and AChE leading to decreased ACh concentrations. In turn, ACh signaling leads to activation of VEGF and AKT and blocking of caspase‐3.
Stuti Goel, Caroline Wozniak, Aya Sabri, Ben Haddad, Brooke Lopo, Alvaro Cobos, Sarah Sarofim, Jeffrey Guthrie, Deborah Heyl, Hedeel Guy Evans   +9 more
wiley   +1 more source

STEROID EXCRETION IN A CASE OF ADRENOCORTICAL CARCINOMA

, 1950
H. Hirschmann, Frieda B. Hirschmann
openalex   +1 more source

Urinary Sodium and Potassium Excretion and CKD Progression.

Journal of the American Society of Nephrology, 2016
Jiang He, K. Mills, L. Appel, Wei Yang, Jing Chen, Belinda T. Lee, S. Rosas, Anna C. Porter, Gail K. Makos, M. Weir, L. Hamm, J. Kusek   +11 more
semanticscholar   +1 more source

Overexpression of CDT1 inhibits cell cycle progression at S phase by interacting with the mini‐chromosome maintenance complex and causes DNA damage

FEBS Open Bio, EarlyView.
CDT1 is an essential protein for DNA replication licensing that loads the MCM complex, the eukaryotic replicative DNA helicase, onto replication origins. Overexpression of CDT1 induces cell cycle arrest at the S phase. Here we showed CDT1 inhibits the progression of replication forks by interacting with the MCM complex, leading to the stalling and ...
Takashi Tsuyama, Nonoka Takayama, Rina Tanaka, Yuuki Arai, Yohko Yamaguchi, Yuko Nawata, Yutaro Azuma, Shusuke Tada   +7 more
wiley   +1 more source