Results 181 to 190 of about 1,227,011 (269)
Medicaid Expansion and Severe Maternal Morbidity. [PDF]
Guernsey EK +6 more
europepmc +1 more source
This study aimed to evaluate the prognostic value of ELN2017 in predicting survival outcomes and to assess the impact of clinical and molecular factors such as age, FLT3 and NPM1 mutations, and allogeneic hematopoietic stem cell transplantation (allo‐HSCT).
Mobina Shrestha +4 more
wiley +1 more source
Total Cranial Vault Remodeling Using Multidirectional Cranial Distraction Osteogenesis in Craniosynostosis. [PDF]
Sunaga A +6 more
europepmc +1 more source
Expansions in Eigenfunctionals
openaire +2 more sources
ABSTRACT As global populations age, cancer is increasingly becoming a leading cause of morbidity and mortality among older adults, particularly in low‐ and middle‐income countries (LMICs). Despite accounting for the majority of new cancer cases and deaths, older individuals remain underrepresented in cancer research, clinical guidelines, and health ...
Ibrahim Bidemi Abdullateef +2 more
wiley +1 more source
Multi-Parameter Collaborative Optimization of Foamed Asphalt Cold Recycling Mixture Properties. [PDF]
Qiu W, Li B, Song Z, Zou X, Hu M, Bi Y.
europepmc +1 more source
Aging Is a Key Driver for Adult Acute Myeloid Leukemia
Acute myeloid leukemia (AML) is a classical age‐related hematologic malignancy, and a key driver of AML is aging, which profoundly regulates intrinsic factors such as genomic instability, epigenetic reprogramming, and metabolic dysregulation, and alters bone marrow microenvironment.
Rong Yin, Haojian Zhang
wiley +1 more source
Rapid Expansion of the Eastern China Lineage (Haplotype P) of <i>Phragmites australis</i> in Caohai Lake, Guizhou, China. [PDF]
Li W +6 more
europepmc +1 more source
Mutant NPM1 in Acute Myeloid Leukemia Initiation and Maintenance
NPM1 mutations drive acute myeloid leukemia by acting as neomorphic transcriptional regulators that cooperate with Menin–MLL and XPO1 to sustain HOX/MEIS1 expression and block differentiation. Targeting these mutant‐specific transcriptional dependencies provides a rational therapeutic strategy for NPM1‐mutated AML.
Yanan Jiang +3 more
wiley +1 more source

