Results 241 to 250 of about 10,192,906 (282)
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Factor X Deficiency and Systemic Amyloidosis
New England Journal of Medicine, 1977The association of an isolated acquired factor X (Stuart-factor) deficiency and systemic acquired amyloidosis has been sporadically reported over the last 15 years,1 , 2 and over two thirds of the ...
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A NEW VARIANT OF FACTOR X DEFICIENCY (FACTOR X ROMA).
Thrombosis and Haemostasis, 1987A 13 years-old-girl was admitted in our Hospital for a large muscle hematoma of left psoas. At age 3 she had a severe tonsillar bleeding following angina. Afterwards she suffered from easy bruisability and recurrent epistaxis. Prothrombin time (PT) was slightly prolonged (14.7 sec, control 12 sec, INR 1.5), while a more marked prolongation in aPTT was ...
V De Stefano +3 more
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Norethynodrel in The Treatment of Factor X Deficiency
Archives of Internal Medicine, 1964The demonstrated improvement of factor X (Prower-Stuart factor) deficiency during pregnancy, 1 and the further demonstration by Alexander and Pechet 2 of increased clotting factors during pregnancy, has prompted the use of norethynodrel, a progestational agent, in the control of this clotting disorder. The case to be reported has been the subject of a
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Replacement therapy for congenital Factor X deficiency
Transfusion, 1985We studied a young woman with severe (< 1%) congenital factor X deficiency during a 2‐year period in order to document the levels of factor X required to provide hemostasis for vaginal bleeding, epistaxis, and hemarthroses, as well as during surgery.
R D, Knight, C F, Barr, B M, Alving
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Mechanism of Factor X Deficiency in Systemic Amyloidosis
New England Journal of Medicine, 1981SOME patients with primary (immunoglobulintype) amyloidosis acquire a coagulation disorder characterized by factor X deficiency1 2 3 4 5 6 7 8 9 10 11 12 13 and occasionally factor IX deficiency.12...
B, Furie +3 more
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Factor X deficiency presenting as a pseudotumor
Journal of Neurosurgery: Pediatrics, 2006The authors report their experience in successfully treating a 15-week-old child who became comatose following a spontaneous intracerebral hemorrhage. It was initially believed that a tumor in the right frontal lobe caused the hemorrhage. Coagulation studies revealed abnormal results on presentation, and the problem was only partially corrected after ...
Jecko V, Thachil +6 more
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JAMA, 1974
Isolated factor X deficiency in association with abnormal plasma antithrombin activity was recognized in a 59-year-old man with amyloidosis and signs and symptoms of calf pain suggestive of thrombophlebitis. Venographic studies excluded venous thrombosis, and, despite major coagulation abnormalities, there was no hemorrhagic tendency.
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Isolated factor X deficiency in association with abnormal plasma antithrombin activity was recognized in a 59-year-old man with amyloidosis and signs and symptoms of calf pain suggestive of thrombophlebitis. Venographic studies excluded venous thrombosis, and, despite major coagulation abnormalities, there was no hemorrhagic tendency.
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Treatment of Amyloidosis Associated Factor X Deficiency
Thrombosis and Haemostasis, 1976SummaryThis is the tenth patient in thirteen years to be reported with the findings of an isolated factor X deficiency associated with primary amyloidosis. A favorable response to factor IX concentrate was manifested by temporary clinical and laboratory correction of her diathesis.
J A, Spero +3 more
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Factor‐X deficiency in amyloidosis: A critical review
American Journal of Hematology, 1981AbstractThirty cases of amyloidosis with factor‐X deficiency, including six of our own, were reviewed. Modest deficiency of factor X was often associated with severe bleeding. In many of the cases, clinical bleeding could not be accounted for by deficiency of factor X alone. Other hemostatic defects were found in these patients and probably contributed
P R, Greipp, R A, Kyle, E J, Bowie
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Systemic Amyloidosis Associated With Factor X Deficiency
Acta Pathologica Japonica, 1984An autopsy case of amyloidosis associated with factor X deficiency is reported. The patient showed a markedly decreased level of factor X (9% normal) and an extremely shortened half‐life of intravenously infused factor X. Amyloid deposition was present in most of the visceral organs with special involvement of the liver and spleen.
H, Shibuya +4 more
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