Results 121 to 130 of about 70,491 (173)
PanERBB CAR T-cells: Shifting gears toward a cure for fanconi anemia head/neck cancers. [PDF]
Mol Ther OncolWiek C +5 more
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Retraction Note: RNA interferences targeting the Fanconi anemia/BRCA pathway upstream genes reverse cisplatin resistance in drug-resistant lung cancer cells. [PDF]
J Biomed SciDai CH +5 more
europepmc +1 more source
Head and Neck Cancer in Fanconi Anemia: Clinical Challenges and Molecular Insights into a DNA Repair Disorder. [PDF]
Cancers (Basel)Choi J, Jung M.
europepmc +1 more source
Statement of Retraction: A novel role for fanconi anemia (FA) pathway effector protein FANCD2 in cell cycle progression of untransformed primary human cells. [PDF]
Cell Cycleeuropepmc +1 more source
Fanconi's anemia and malignancies [PDF]
American Journal of Hematology, 1996 Patients with Fanconi's anemia (FA) are at a high risk for development of malignancies. It is well-known that leukemia occurs in approximately 10% of cases, with increasing risk with age. Less commonly recognized is the risk for myelodysplastic syndromes (approximately 5%); the relationship between myelodysplasia and evolution to leukemia remains ...
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Seminars in Hematology, 2006
Fanconi anemia (FA) is a rare hereditary disease characterized by bone marrow failure and developmental anomalies; a high incidence of myelodysplasia (MDS), acute nonlymphocytic leukemia (AML), and solid tumors; and cellular hypersensitivity to cross-linking agents. The genetic basis of FA is mutations in any one of the known FA genes.
Grover C, Bagby, Blanche P, Alter
+5 more sources
Fanconi anemia (FA) is a rare hereditary disease characterized by bone marrow failure and developmental anomalies; a high incidence of myelodysplasia (MDS), acute nonlymphocytic leukemia (AML), and solid tumors; and cellular hypersensitivity to cross-linking agents. The genetic basis of FA is mutations in any one of the known FA genes.
Grover C, Bagby, Blanche P, Alter
+5 more sources
Blood, 2003
Three separate and complementary reports recently described the leukemia and solid tumor experience in cohorts of patients with Fanconi anemia (FA).[1-3][1] Here we examine the similarities and differences of these reports (Table[1][2]) in order to synthesize the most current evidence for ...
Philip S. Rosenberg
exaly +3 more sources
Three separate and complementary reports recently described the leukemia and solid tumor experience in cohorts of patients with Fanconi anemia (FA).[1-3][1] Here we examine the similarities and differences of these reports (Table[1][2]) in order to synthesize the most current evidence for ...
Philip S. Rosenberg
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Pediatrics, 1980
We report a 10-year-old boy who has Fanconi's anemia and was admitted because of acute hemiplegia of the left side. Internal carotid arteriography disclosed a moyamoya cerebrovascular pattern on the right side. Although this condition may be acquired, it is suggested that in this case the moyamoya might pertain to the array of congenital malformations ...
Moshe Berant, M Berant
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We report a 10-year-old boy who has Fanconi's anemia and was admitted because of acute hemiplegia of the left side. Internal carotid arteriography disclosed a moyamoya cerebrovascular pattern on the right side. Although this condition may be acquired, it is suggested that in this case the moyamoya might pertain to the array of congenital malformations ...
Moshe Berant, M Berant
exaly +3 more sources
New England Journal of Medicine, 1994
To the Editor: We have several comments on the Image in Clinical Medicine entitled “Some Features of Fanconi's Anemia” (Oct. 14 issue)1.
P F, Giampietro +2 more
openaire +2 more sources
To the Editor: We have several comments on the Image in Clinical Medicine entitled “Some Features of Fanconi's Anemia” (Oct. 14 issue)1.
P F, Giampietro +2 more
openaire +2 more sources
Current Opinion in Pediatrics, 1998
Until recently, it had been thought that Fanconi anemia patients were distributed into five complementation groups. However, evidence now points to the existence of three new complementation groups, making the genetic basis of the disease more complicated than anticipated.
M, Carreau, M, Buchwald
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Until recently, it had been thought that Fanconi anemia patients were distributed into five complementation groups. However, evidence now points to the existence of three new complementation groups, making the genetic basis of the disease more complicated than anticipated.
M, Carreau, M, Buchwald
openaire +2 more sources