Results 261 to 270 of about 232,776 (304)

Targeted Deletion of Fibroblast Growth Factor 23 Rescues Metabolic Dysregulation of Diet-induced Obesity in Female Mice.

open access: yesEndocrinology
Park MY   +13 more
europepmc   +1 more source

Fibroblast growth factor 23

Annals of Clinical Biochemistry: International Journal of Laboratory Medicine, 2013
There is growing interest in the role of fibroblast growth factor 23 (FGF23) in various diseases of disordered mineral metabolism. In chronic kidney disease (CKD), where biochemical evidence of mineral disturbances is especially common, FGF23 measurement has been advocated as an early and sensitive marker for CKD-related bone disease. In this setting,
Edward R, Smith   +2 more
openaire   +2 more sources

Determination of fibroblast growth factor 23

Annals of Clinical Biochemistry: International Journal of Laboratory Medicine, 2009
Background Fibroblast growth factor 23 (FGF-23) is a recently discovered hormone, which plays a key role in phosphate regulation. To investigate whether FGF-23 can be determined reliably, we validated the three available FGF-23 assays. Methods Currently, two intact FGF-23 assays (Kainos; Immutopics) and one C-terminal FGF-23 assay (Immutopics) are ...
Heijboer, Annemieke C.   +6 more
openaire   +3 more sources

Fibroblast Growth Factor 23 and Its Receptors

Therapeutic Apheresis and Dialysis, 2005
Abstract:  Fibroblast growth factor 23 (FGF23) is a circulating factor that plays critical roles in phosphate and vitamin D metabolism, as evidenced by the fact that FGF23 missense mutations cause autosomal dominant hypophosphatemic rickets (ADHR). Autosomal dominant hypophosphatemic rickets is characterized by hypophosphatemia with inappropriately ...
Xijie, Yu, Kenneth E, White
openaire   +2 more sources

Inherited fibroblast growth factor 23 excess

Best Practice & Research Clinical Endocrinology & Metabolism
Syndromes of inherited fibroblast growth factor 23 (FGF-23) excess encompass a wide spectrum that includes X-linked hypophosphataemia (XLH), autosomal dominant and recessive forms of rickets as well as various syndromic conditions namely fibrous dysplasia/McCune Albright syndrome, osteoglophonic dysplasia, Jansen's chondrodysplasia and cutaneous ...
Kripa Elizabeth, Cherian   +1 more
openaire   +2 more sources

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