Finger osteochondroma presenting as non-traumatic mallet finger in pediatric: A case report. [PDF]
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Visualization of Atherosclerotic Plaques Paired with Joheksol 350 (Omnipaque). [PDF]
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Inflammatory Pathways in Coronary Artery Disease: Which Ones to Target for Secondary Prevention? [PDF]
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The value of contrast-enhanced ultrasound in assessing carotid plaque vulnerability and predicting stroke risk. [PDF]
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Inflammation mediates the relationship between cardiometabolic index and vulnerable plaque in patients with acute coronary syndrome. [PDF]
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Microcalcifications, Their Genesis, Growth, and Biomechanical Stability in Fibrous Cap Rupture
Advances in Experimental Medicine and Biology, 2018For many decades, cardiovascular calcification has been considered as a passive process, accompanying atheroma progression, correlated with plaque burden, and apparently without a major role on plaque vulnerability. Clinical and pathological analyses have previously focused on the total amount of calcification (calcified area in a whole atheroma cross ...
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Three‐dimensional imaging of fibrous cap by frequency‐domain optical coherence tomography
Catheterization and Cardiovascular Interventions, 2012Histopathological data correlated the presence of thin‐cap fibroatheroma (TCFA) with adverse cardiovascular events. Intravascular optical coherence tomography (iOCT) is currently the only imaging modality with sufficiently high axial resolution (∼15 μm) that allows direct fibrous‐cap (FC) assessment in vivo.
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Semi-automated Quantification of Fibrous Cap Thickness in Intracoronary Optical Coherence Tomography
International Conference on Information Processing in Computer-Assisted Interventions, 2014Acute coronary syndrome represents a leading cause of death. Events are triggered by rupture of atheromatic plaques, as a result of disruption of the overlying fibrous cap. Pathological studies have shown that cap thickness is a critical component of plaque stability.
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