Results 111 to 120 of about 247,844 (268)

Uniform boundary controllability of a discrete 1-D Schrodinger equation

open access: yesKarpatsʹkì Matematičnì Publìkacìï, 2015
In this paper we study the controllability of a finite dimensional system obtained by discretizing in space and time the linear 1-D Schrodinger equation with a boundary control.
Z. Hajjej, M. Balegh
doaj   +1 more source

Heterozygous loss‐of‐function alleles associate the conserved 3′‐5′ exoribonuclease EXOSC10 with hypersensitivity to the anticancer drug 5‐fluorouracil

open access: yesMolecular Oncology, EarlyView.
EXOSC10, an essential nuclear RNA exosome‐associated 3′‐5′ exoribonuclease, is inhibited by the anticancer drug 5‐fluorouracil (5‐FU), and EXOSC10 depletion increases 5‐FU sensitivity. The colon‐cancer variant EXOSC10S402T, located in a proteolysis motif, is stable and nuclear but nonfunctional in vivo.
Radhika Sain   +10 more
wiley   +1 more source

Robust H∞ filtering for discrete nonlinear stochastic systems with time-varying delay

open access: yes, 2008
This is the postprint version of the article. The official published version can be accessed from the link below - © 2007 Elsevier IncIn this paper, we are concerned with the robust H∞ filtering problem for a class of nonlinear discrete time-delay ...
Liu, Y   +8 more
core   +1 more source

Multi-Modal Filtering for Non-linear Estimation [PDF]

open access: yes, 2014
16.01.14 KB. Ok to add to spiral, authors retain copyrightMulti-modal densities appear frequently in time series and practical applications. However, they are not well represented by common state estimators, such as the Extended Kalman Filter and the ...
Deisenroth, Marc P   +11 more
core   +1 more source

Cell‐cycle‐specific lesion evolution rather than inhibition of double‐strand‐break repair underpins cisplatin radiosensitization

open access: yesMolecular Oncology, EarlyView.
We analyze cisplatin–DNA adducts (CDAs) and double‐strand breaks (DSBs) in a cell‐cycle‐dependent manner. We find that CDAs form similarly across all cell cycle phases. DSBs arise only in S‐phase. CDAs might not directly impair DSB repair, but S‐phase DSB lesions evolve in the presence of CDAs and disrupt repair in G2, also causing radiosensitization ...
Ye Qiu   +10 more
wiley   +1 more source

Optimal HP filtering for South Africa [PDF]

open access: yes
Among the various methods used to identify the business cycle from aggregate data, the Hodrick-Prescott filter has become an industry standard – it ‘identifies’ the business cycle by removing low-frequency information, thereby smoothing the data.
Leon du Toit
core  

A parallel Kalman filter via the square root Kalman filtering [PDF]

open access: yes, 1993
A parallel algorithm for Kalman filtering with contaminated observations is developed. Theı parallel implementation is based on the square root version of the Kalman filter (see [3]).
Romera, Rosario, Cipra, Tomas
core  

Noise Reduction on G‐Buffers for Monte Carlo Filtering

open access: yes, 2017
We propose a novel pre‐filtering method that reduces the noise introduced by depth‐of‐field and motion blur effects in geometric buffers (G‐buffers) such as texture, normal and depth images.
Mitchell, Kenny   +3 more
core   +1 more source

Longitudinal circulating tumor DNA profiling in patients with advanced endometrial cancer using an off‐the‐shelf targeted NGS panel

open access: yesMolecular Oncology, EarlyView.
Intratumour heterogeneity complicates precision management of advanced endometrial cancer. Circulating tumor DNA (ctDNA) offers a minimally invasive strategy to capture tumor evolution and therapeutic resistance. Here, we compare tumor‐agnostic NGS with tumor‐informed ddPCR, outlining their relative sensitivity, concordance, and clinical implications ...
Carlos Casas‐Arozamena   +15 more
wiley   +1 more source

Interaction of HS1BP3 with cortactin modulates TKS5 localisation, cell secretion and cancer malignancy

open access: yesMolecular Oncology, EarlyView.
Here, we demonstrate that HS1BP3 interacts with Cortactin through a proline‐rich region (PRR3.1) and show that this interaction, and HS1BP3 itself, promote cancer cell proliferation and invasion. Inhibition of this interaction leads to build‐up of TKS5 in multivesicular endosomes and altered secretion of CD63 and CD9, providing an explanation for the ...
Arja Arnesen Løchen   +9 more
wiley   +1 more source

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