Results 151 to 160 of about 22,351 (243)

Investigation of fingolimod-induced lymphocyte sequestration on inflammatory response and neurological damages after cardiac arrest

open access: yesIntensive Care Medicine Experimental
Background A sepsis-like syndrome is known to occur after cardiac arrest, leading to cerebral infiltration by white blood cells (WBC). We hypothesized that pharmacological sequestration of WBC, and more specifically lymphocytes within lymphoid tissues ...
Yara Abi Zeid Daou   +11 more
doaj   +1 more source

Rituximab versus fingolimod after natalizumab in multiple sclerosis patients

open access: yes, 2016
Objective: Many JC virus antibody-positive relapsing-remitting multiple sclerosis (RRMS) patients who are stable on natalizumab switch to other therapies to avoid progressive multifocal leukoencephalopathy.Methods: We compared outcomes for all RRMS ...
Frisell, T.   +35 more
core   +1 more source

Oral fingolimod or intramuscular interferon for relapsing multiple sclerosis.

open access: yesNew England Journal of Medicine, 2010
Jeffrey A. Cohen   +16 more
semanticscholar   +1 more source

Cladribine versus fingolimod, natalizumab and interferon β for multiple sclerosis

open access: yes, 2018
Objective: This propensity score–matched analysis from MSBase compared the effectiveness of cladribine with interferon β, fingolimod or natalizumab.
Horakova D.   +27 more
core   +1 more source

Fingolimod increases cellular resistance to HIV-1 infection and limits viral reservoir size in peripheral CD4+ T-cells. [PDF]

open access: yesPLoS Pathog
Moraga E   +12 more
europepmc   +1 more source

Chronic HPV associated lesions associated with Fingolimod use

open access: yes, 2018
Background: Fingolimod is a an effective immunosuppressive therapy used to reduce relapse rates in relapsing remitting MS. Fingolimod is a sphingosine 1-phosphate (S1-P) analogue and has antagonistic effects on S1-P receptors causing in S1-P receptor ...
Kermode, A.G., Triplett, J.D.
core  

LncRNA AFAP1-AS1 mediates therapy-dependent expression of CCL5, CXCL10 and MMP9 in multiple sclerosis. [PDF]

open access: yesBiochem Biophys Rep
Hegazy LN   +4 more
europepmc   +1 more source

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