Results 21 to 30 of about 19,821 (199)

Hereditary fructose intolerance. [PDF]

open access: yesMaandschrift voor kindergeneeskunde, 1998
Hereditary fructose intolerance (HFI, OMIM 22960), caused by catalytic deficiency of aldolase B (fructose-1,6-bisphosphate aldolase, EC 4.1.2.13), is a recessively inherited condition in which affected homozygotes develop hypoglycaemic and severe abdominal symptoms after taking foods containing fructose and cognate sugars.
E R, FROESCH   +3 more
  +8 more sources

Fructose transporters GLUT5 and GLUT2 expression in adult patients with fructose intolerance [PDF]

open access: yesUnited European Gastroenterology Journal, 2014
Clive H Wilder-Smith, Ronaldo P Ferraris
exaly   +2 more sources

Gene variants of the SLC2A5 gene encoding GLUT5, the major fructose transporter, do not contribute to clinical presentation of acquired fructose malabsorption

open access: yesBMC Gastroenterology, 2022
Background While role of ALDOB-related gene variants for hereditary fructose intolerance is well established, contribution of gene variants for acquired fructose malabsorption (e.g. SLC2A5, GLUT5) is not well understood.
Irina Taneva   +6 more
doaj   +1 more source

Beneficial effects of calcitriol on hypertension, glucose intolerance, impairment of endothelium-dependent vascular relaxation, and visceral adiposity in fructose-fed hypertensive rats. [PDF]

open access: yesPLoS ONE, 2015
Besides regulating calcium homeostasis, the effects of vitamin D on vascular tone and metabolic disturbances remain scarce in the literature despite an increase intake with high-fructose corn syrup worldwide. We investigated the effects of calcitriol, an
Chu-Lin Chou   +3 more
doaj   +1 more source

Case report: Carbohydrate malabsorption in inpatients with anorexia nervosa

open access: yesFrontiers in Psychiatry, 2022
BackgroundGastrointestinal (GI) complaints are frequently observed in patients who suffer from anorexia nervosa (AN). These symptoms may hamper treatment and weight regain and are often perceived as the cause, not the consequence, of the disease.
Patrizia Buck   +6 more
doaj   +1 more source

A fruktóz és a fruktózintolerancia | Fructose and fructose intolerance [PDF]

open access: yes, 2016
Absztrakt A fruktóz gyümölcsökben és zöldségekben természetesen előforduló monoszacharid. Az utóbbi évtizedekben világszerte jelentősen megnövekedett a fruktóz bevitele, elsősorban az ...
Buzás, György Miklós
core   +1 more source

Exercise Training Prevents Cardiovascular Derangements Induced by Fructose Overload in Developing Rats. [PDF]

open access: yesPLoS ONE, 2016
The risks of chronic diseases associated with the increasing consumption of fructose-laden foods are amplified by the lack of regular physical activity and have become a serious public health issue worldwide.
Daniela Farah   +11 more
doaj   +1 more source

Glucose but Not Fructose Alters the Intestinal Paracellular Permeability in Association With Gut Inflammation and Dysbiosis in Mice

open access: yesFrontiers in Immunology, 2021
A causal correlation between the metabolic disorders associated with sugar intake and disruption of the gastrointestinal (GI) homeostasis has been suggested, but the underlying mechanisms remain unclear.
Xufei Zhang   +6 more
doaj   +1 more source

Inborn Errors of Fructose Metabolism. What Can We Learn from Them? [PDF]

open access: yes, 2017
Fructose is one of the main sweetening agents in the human diet and its ingestion is increasing globally. Dietary sugar has particular effects on those whose capacity to metabolize fructose is limited.
Tran, C.
core   +2 more sources

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