Results 241 to 250 of about 352,265 (309)

Recent Advances in Targeted Therapies for Adult Gliomas. [PDF]

Curr Neurol Neurosci Rep
Lasica AB, Tang AR, Iqbal A, Nakhate V, Wen PY.   +4 more
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Enhancing clinical insights in glioma grading using Bayesian Optimization and Explainable AI. [PDF]

J Genet Eng Biotechnol
Elsayad AM, Elsayad OA.
europepmc   +1 more source

Triple-Targeted DNA Nanozyme Executes Disulfidptosis for Glioma Elimination. [PDF]

Theranostics
Wu T, Li L, Zhang S, Zhang G, Li C, Mei J, Chen X, Cheng J.   +7 more
europepmc   +1 more source

Redefining the immune microenvironment of gliomas in the era of single-cell genomics. [PDF]

Neurooncol Adv
Gonzalez Castro LN, Kumar VS, Suva ML, Miller TE.   +3 more
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Tumor-suppressive role of miR-216b impacts cell cycle regulation in high-grade glioma. [PDF]

Acta Neuropathol Commun
Lang A, Buerger S, Mager L, Erjavc L, Kiesel B, Madlener S, Jaunecker CN, Pacher L, Robert FC, Krausgruber L, Nascimento A, Machreich S, Ricken G, Gabler-Pamer L, Laemmerer A, Spiegl-Kreinecker S, Hackl M, Pultar M, Reisecker J, Bruckner K, Neudert B, Muellauer L, Kirchhofer D, Mischkulnig M, Berghoff AS, Woehrer A, Senfter D, Roessler K, Roetzer-Pejrimovsky T, Berger W, Hainfellner JA, Hoeftberger R, Widhalm G, Erhart F, Lötsch-Gojo D.   +34 more
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Metabolic dependencies and neural progenitor dysregulation: driving forces in paediatric high-grade glioma development. [PDF]

Cancer Metastasis Rev
Lee YHG, Tsoli M, Shi YC, Ip CK, Ziegler D.   +4 more
europepmc   +1 more source

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Malignant Glioma

2023
This chapter provides a comprehensive overview of malignant gliomas, the most common primary brain tumor in adults. These tumors are varied in their cellular origin, genetic profile, and morphology under the microscope, but together they share some of the most dismal prognoses of all neoplasms in the body.
Linda M, Wang, Zachary K, Englander, Michael L, Miller, Jeffrey N, Bruce   +3 more
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Multiple gliomas

Voprosy neirokhirurgii imeni N.N. Burdenko, 2022
The authors present 2 patients. One of them had typical multifocal primary multiple synchronous wild-type IDH1/2 glioblastoma subtype RTK1, chromosome 7 duplication, homozygous CDKN2A deletion and chromosome 10 deletion. In another patient, the nature of tumors remains debatable.
M.V. Ryzhova, S.A. Galstyan, E.N. Telysheva, E.I. Petrova, G.L. Kobyakov, A.I. Khodzhaev, S.A. Maryashev   +6 more
openaire   +2 more sources