Results 171 to 180 of about 21,381 (225)
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IgA-Associated Glomerulonephritis
Annual Review of Medicine, 1977In summary, IgA-associated glomerulonephritis is an interesting clinical problem. The immunohistochemical identification of renal IgA deposits is the sine qua non of its diagnosis, although most of the patients reported have had hematuric syndromes, particularly recurrent gross hematuria. The importance of this immunopathologic entity devolves from the
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International Urology and Nephrology, 1979
Renal biopsy specimens from 204 patients with glomerulonephritis or nephrotic syndrome have been studied. In ten of the patients not suffering from acute poststreptococcal glomerulonephritis, systemic lupus erythematosus or Schönlein-Henoch syndrome, diffuse, selective mesangial IgA deposition was observed. Clinically, persistent microscopic haematuria,
J, Nagy +5 more
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Renal biopsy specimens from 204 patients with glomerulonephritis or nephrotic syndrome have been studied. In ten of the patients not suffering from acute poststreptococcal glomerulonephritis, systemic lupus erythematosus or Schönlein-Henoch syndrome, diffuse, selective mesangial IgA deposition was observed. Clinically, persistent microscopic haematuria,
J, Nagy +5 more
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IgA, Glomerulonephritis and Liver Disease
Internal Medicine Journal, 1981Abstract: Data from our Unit suggest that soluble immune complexes (IC) are responsible for the mesangial deposits of IgA and C3 in patients with IgA nephropathy. These IC are of intermediate size (9–17S) and contain IgA, IgG and less commonly IgM.
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Chronic glomerulonephritis with massive IgA deposits
International Urology and Nephrology, 1985A total of 234 patients with chronic glomerulonephritis, detected on the basis of clinical, immunological and pathomorphological criteria, have been examined. A group of 23 chronic glomerulonephritis patients with massive deposits of immunoglobulin A found by immunofluorescent examination, has been separated on the basis of immunohistochemical studies.
A, Steciwko +3 more
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A Proliferative Glomerulonephritis Secondary to a Monoclonal IgA
American Journal of Kidney Diseases, 2006A distinct entity mimicking immune-complex-mediated glomerulonephritis characterized by a proliferative glomerulonephritis with monoclonal immunoglobulin G (IgG) deposits recently was described. We now report a case of a 35-year-old woman who presented with sudden onset of edema, proteinuria, hematuria, and hypertension.
Sandra M, Soares +4 more
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La Revue du praticien, 1992
Primary IgA nephritis is the most frequent nephritis and represent half of the glomerular diseases. Its incidence is 2,7/10(5) inhabitants. The diagnosis is established on a renal biopsy, which shows typical IgA mesangial deposits: granular, predominant, and diffuse. A central pathogenic role is played by the IgA immune system, both from the mucosa and
E, Alamartine, F, Berthoux
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Primary IgA nephritis is the most frequent nephritis and represent half of the glomerular diseases. Its incidence is 2,7/10(5) inhabitants. The diagnosis is established on a renal biopsy, which shows typical IgA mesangial deposits: granular, predominant, and diffuse. A central pathogenic role is played by the IgA immune system, both from the mucosa and
E, Alamartine, F, Berthoux
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IgA-dominant glomerulonephritis associated with hepatitis A
Clinical Nephrology, 2004Unlike hepatitis B and C, renal involvement has been extremely uncommon in patients with hepatitis Avirus (HAV) infection. Nephrotic syndrome has been documented as a rare complication in association with HAV infection. In this report, we describe a patient with serologically documented HAV infection, who presented with nephrotic syndrome.
S R, Cheema +4 more
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Lymphocyte subpopulations in mesangial IgA glomerulonephritis
International Urology and Nephrology, 1988Lymphocyte subpopulations in thirty patients with IgA glomerulonephritis and in twenty-four healthy persons were identified using specific monoclonal antibodies. Decreased OKT11+ cells (37.83 +/- 13.34, p less than 0.01) without any changes of OKT4+ cells, but increased OKT8+ cells (25.89 +/- 6.70, p less than 0.01) in patients with IgA ...
J P, Altunkova +3 more
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[Tubular proteinuria in IgA glomerulonephritis].
Orvosi hetilap, 1987In the renal biopsy samples of some patients with IgA glomerulonephritis (IgA GN), tubulointerstitial changes and a significant correlation between these changes and the serum creatinine levels had been observed earlier. In order to get an insight into the function of the tubules, 45 patients with IgA GN have been examined for proteinuria with special ...
J, Nagy +3 more
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[Mesangial IgA-glomerulonephritis].
Deutsche medizinische Wochenschrift (1946), 1983IgA-glomerulonephritis represents the most frequent glomerulonephritis (GN; 20%) among our patients. In contrast to data from the literature the prognosis is not benign. Renal insufficiency developed in 17 out of 50 investigated patients within 4 to 96 months, 3 of these patients had to undergo dialysis.
M, Rambausek +6 more
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