Emerging role of the pentose phosphate pathway in hepatocellular carcinoma [PDF]
, 2017 In recent years, there has been a revival of interest in metabolic changes of cancer cells as it has been noticed that malignant transformation and metabolic reprogramming are closely intertwined.
Columbano, Amedeo +2 more
core +2 more sources
Prevalence of glucose-6-phosphate dehydrogenase deficiency and its association with Plasmodium falciparum infection among children in Iganga distric in Uganda [PDF]
, 2014 BACKGROUND: Glucose-6-phosphate dehydrogenase (G6PD) is a metabolic enzyme involved in the pentose phosphate pathway, its especially important in red blood cell metabolism.
Denis Bwayo +3 more
core +1 more source
G6PD Deficiency in Neonatal Jaundice
Van Tıp Dergisi, 2022 INTRODUCTION: Neonatal jaundice is one of the important problems encountered by the newborn in the first week after birth, the etiology of which is very diverse and sometimes no cause can be detected.
Altay Babacan +2 more
doaj +1 more source
Nutritional status of young children with inherited blood disorders in western Kenya. [PDF]
, 2014 To determine the association between a range of inherited blood disorders and indicators of poor nutrition, we analyzed data from a population-based, cross-sectional survey of 882 children 6–35 months of age in western Kenya.
Ruth, LJ +4 more
core +1 more source
MOLECULAR BASIS OF G6PD DEFICIENCY: CURRENT STATUS AND ITS PERSPECTIVE [PDF]
Acta Medica Iranica, 2008 Glucose-6-phosphate dehydrogenase is an essential enzyme to cell growth. Its deficiency of enzyme plays an important role in senescence and death signaling.
M. R. Noori-Daloii, M. Daneshpajooh
doaj +2 more sources
Glucose-6-Phosphate Dehydrogenase Deficiency and Neonatal Hyperbilirubinemia [PDF]
Iranian Journal of Neonatology, 2015 Background: Jaundice is affecting over 60-80 percent of neonates in the first week of life. Glucose-6-phosphate dehydrogenase (G6PD) deficiency, which is an important cause of pathologic hyperbilirubinemia, can lead to hemolytic anemia, jaundice and ...
Ezzat Khodashenas +4 more
doaj
Using in silico models to simulate dual perturbation experiments: procedure development and interpretation of outcomes. [PDF]
, 2009 BackgroundA growing number of realistic in silico models of metabolic functions are being formulated and can serve as 'dry lab' platforms to prototype and simulate experiments before they are performed.
Jamshidi, Neema, Palsson, Bernhard O
core +3 more sources
Molecular Oncology, EarlyView.Meta‐transcriptome analysis identified FGF19 as a peptide enteroendocrine hormone associated with colorectal cancer prognosis. In vivo xenograft models showed release of FGF19 into the blood at levels that correlated with tumor volumes. Tumoral‐FGF19 altered murine liver metabolism through FGFR4, thereby reducing bile acid synthesis and increasing ...
Jordan M. Beardsley +5 more
wiley +1 more source
Glucose-6-phosphate Dehydrogenase Deficiency
Clinical Pediatric Hematology-Oncology, 2007 Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most common enzyme disorder. There are more than 400 million people worldwide with G6PD deficiency, and its distribution is similar to that of malaria. G6PD deficiency is an X-linked recessive disorder. Most patients with G6PD deficiency may be asymptomatic throughout their lives.
openaire +1 more source
Glucose 6 phosphate dehydrogenase deficiency in Malta : a preliminary study [PDF]
, 1968 It has been suggested that glucose-6-phosphate dehydrogenase deficiency (G6PD) affects more than 100 million people of all races throughout the world. The special relevance of this problem to Malta can readily be appreciated when it is remembered that ...
Cauchi, Maurice N.
core